Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt.
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Dinosaurs and ancient civilizations: reflections on the treatment of cancerChaperone-targeting cytotoxin and endoplasmic reticulum stress-inducing drug synergize to kill cancer cellsModulation of the Akt pathway reveals a novel link with PERK/eIF2α, which is relevant during hypoxiaSynergistic antitumor activities of docetaxel and octreotide associated with apoptotic-upregulation in castration-resistant prostate cancerBufalin Reverses Resistance to Sorafenib by Inhibiting Akt Activation in Hepatocellular Carcinoma: The Role of Endoplasmic Reticulum StressTamoxifen enhances the cytotoxic effects of nelfinavir in breast cancer cells.Ets-1 mediates upregulation of Mcl-1 downstream of XBP-1 in human melanoma cells upon ER stress.The interconnectedness of cancer cell signaling.RIPK1 regulates survival of human melanoma cells upon endoplasmic reticulum stress through autophagy.Salubrinal-Mediated Upregulation of eIF2α Phosphorylation Increases Doxorubicin Sensitivity in MCF-7/ADR Cells.Upregulated ATF6 contributes to chronic intermittent hypoxia-afforded protection against myocardial ischemia/reperfusion injurySuppression of antifolate resistance by targeting the myosin Va trafficking pathway in melanoma.Vemurafenib potently induces endoplasmic reticulum stress-mediated apoptosis in BRAFV600E melanoma cells.1,3-Bis(3,5-dichlorophenyl) urea compound 'COH-SR4' inhibits proliferation and activates apoptosis in melanoma.Human melanoma cells under endoplasmic reticulum stress are more susceptible to apoptosis induced by the BH3 mimetic obatoclax.The War on Cancer rages on.IRE1α-XBP1 pathway promotes melanoma progression by regulating IL-6/STAT3 signaling.The role of circadian timing system on drug metabolism and detoxification.Systemic therapy of non-resectable metastatic melanoma.Targeting the hallmarks of cancer with therapy-induced endoplasmic reticulum (ER) stress.Lysosomes and unfolded protein response, determinants of differential resistance of melanoma cells to vinca alkaloids.PI3K/Akt pathway mediates high glucose-induced lipid accumulation in human renal proximal tubular cells via spliced XBP-1.Melatonin sensitizes human hepatoma cells to endoplasmic reticulum stress-induced apoptosis.The unfolded protein response in melanocytes: activation in response to chemical stressors of the endoplasmic reticulum and tyrosinase misfolding.Opa-interacting protein 5 modulates docetaxel-induced cell death via regulation of mitophagy in gastric cancer.The unfolded protein response impacts melanoma progression by enhancing FGF expression and can be antagonized by a chemical chaperone.
P2860
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P2860
Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2009
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Human melanoma cells under end ...... -1-mediated activation of Akt.
@en
Human melanoma cells under end ...... -1-mediated activation of Akt.
@nl
type
label
Human melanoma cells under end ...... -1-mediated activation of Akt.
@en
Human melanoma cells under end ...... -1-mediated activation of Akt.
@nl
prefLabel
Human melanoma cells under end ...... -1-mediated activation of Akt.
@en
Human melanoma cells under end ...... -1-mediated activation of Akt.
@nl
P2093
P2860
P356
P1433
P1476
Human melanoma cells under end ...... -1-mediated activation of Akt.
@en
P2093
Chen Chen Jiang
Peter Hersey
Rick F Thorne
Xu Dong Zhang
P2860
P304
P356
10.1593/NEO.09208
P577
2009-05-01T00:00:00Z