Exendin-4 induced glucagon-like peptide-1 receptor activation reverses behavioral impairments of mild traumatic brain injury in mice - Wikidata - wikimedia - TriplyDB

Exendin-4 induced glucagon-like peptide-1 receptor activation reverses behavioral impairments of mild traumatic brain injury in mice

Exendin-4 induced glucagon-like peptide-1 receptor activation reverses behavioral impairments of mild traumatic brain injury in mice

http://www.wikidata.org/entity/Q37179909

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Hippocampal Neurophysiologic Changes after Mild Traumatic Brain Injury and Potential Neuromodulation Treatment Approaches Exenatide Facilitates Recovery from Oxaliplatin-Induced Peripheral Neuropathy in Rats Oral Administration of Sitagliptin Activates CREB and Is Neuroprotective in Murine Model of Brain Trauma Exendin-4 attenuates blast traumatic brain injury induced cognitive impairments, losses of synaptophysin and in vitro TBI-induced hippocampal cellular degeneration Incretin mimetics as pharmacologic tools to elucidate and as a new drug strategy to treat traumatic brain injury Cognitive impairments accompanying rodent mild traumatic brain injury involve p53-dependent neuronal cell death and are ameliorated by the tetrahydrobenzothiazole PFT-α.Exendin-4 ameliorates traumatic brain injury-induced cognitive impairment in rats.Preservation of the blood brain barrier and cortical neuronal tissue by liraglutide, a long acting glucagon-like-1 analogue, after experimental traumatic brain injury.Liraglutide is neurotrophic and neuroprotective in neuronal cultures and mitigates mild traumatic brain injury in mice.Blast traumatic brain injury-induced cognitive deficits are attenuated by preinjury or postinjury treatment with the glucagon-like peptide-1 receptor agonist, exendin-4.Amelioration of Metabolic Syndrome-Associated Cognitive Impairments in Mice via a Reduction in Dietary Fat Content or Infusion of Non-Diabetic Plasma.Changes in mouse cognition and hippocampal gene expression observed in a mild physical- and blast-traumatic brain injury.Glucose-Dependent Insulinotropic Polypeptide Ameliorates Mild Traumatic Brain Injury-Induced Cognitive and Sensorimotor Deficits and Neuroinflammation in Rats.Dietary resistant starch improves selected brain and behavioral functions in adult and aged rodents.Neuropsychiatric Symptom Modeling in Male and Female C57BL/6J Mice after Experimental Traumatic Brain Injury.Inflammation-sleep interface in brain disease: TNF, insulin, orexin Linking Alzheimer's disease and type 2 diabetes mellitus via aberrant insulin signaling and inflammation.Mild traumatic brain injury-induced hippocampal gene expressions: The identification of target cellular processes for drug development.Neurotrophic and neuroprotective effects of oxyntomodulin in neuronal cells and a rat model of stroke.Novel GLP-1R/GIPR co-agonist "twincretin" is neuroprotective in cell and rodent models of mild traumatic brain injury.Insulin Resistance and Neurodegeneration: Progress Towards the Development of New Therapeutics for Alzheimer's Disease.The Emerging Role of GLP-1 Receptors in DNA Repair: Implications in Neurological Disorders.Insulin-associated neuroinflammatory pathways as therapeutic targets for traumatic brain injury Exendin-4, a glucagon-like peptide-1 receptor agonist, inhibits Aβ25-35-induced apoptosis in PC12 cells by suppressing the expression of endoplasmic reticulum stress-related proteins.Novel pharmaceutical treatments for minimal traumatic brain injury and evaluation of animal models and methodologies supporting their development.Clostridium butyricum exerts a neuroprotective effect in a mouse model of traumatic brain injury via the gut-brain axis.The possible factors affecting microglial activation in cases of obesity with cognitive dysfunction.Exendin-4 attenuates neuronal death via GLP-1R/PI3K/Akt pathway in early brain injury after subarachnoid hemorrhage in rats.Dopamine Release Impairment in Striatum After Different Levels of Cerebral Cortical Fluid Percussion Injury.Are GLP-1 receptor agonists useful against traumatic brain injury?Glucose-Dependent Insulinotropic Polypeptide Mitigates 6-OHDA-Induced Behavioral Impairments in Parkinsonian Rats.Block of A1 astrocyte conversion by microglia is neuroprotective in models of Parkinson's disease

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Exendin-4 induced glucagon-like peptide-1 receptor activation reverses behavioral impairments of mild traumatic brain injury in mice

http://www.wikidata.org/entity/Q37179909

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 15 August 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Exendin-4 induced glucagon-lik ...... traumatic brain injury in mice

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Exendin-4 induced glucagon-lik ...... raumatic brain injury in mice.

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Exendin-4 induced glucagon-lik ...... traumatic brain injury in mice

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Exendin-4 induced glucagon-lik ...... raumatic brain injury in mice.

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Exendin-4 induced glucagon-lik ...... traumatic brain injury in mice

@en

Exendin-4 induced glucagon-lik ...... raumatic brain injury in mice.

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Exendin-4 induced glucagon-lik ...... traumatic brain injury in mice

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Barry J Hoffer

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Chaim G Pick

http://www.w3.org/2001/XMLSchema#string

David Tweedie

http://www.w3.org/2001/XMLSchema#string

Harold W Holloway

http://www.w3.org/2001/XMLSchema#string

Jonathan Miller

http://www.w3.org/2001/XMLSchema#string

Lital Rachmany

http://www.w3.org/2001/XMLSchema#string

Nigel H Greig

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Vardit Rubovitch

http://www.w3.org/2001/XMLSchema#string

Yazhou Li

http://www.w3.org/2001/XMLSchema#string

P2860

Glucagon-like peptide-1 receptor is involved in learning and neuroprotection

Exenatide once weekly versus twice daily for the treatment of type 2 diabetes: a randomised, open-label, non-inferiority study

Exendin-4 ameliorates motor neuron degeneration in cellular and animal models of amyotrophic lateral sclerosis

Tumor necrosis factor-α synthesis inhibitor, 3,6'-dithiothalidomide, reverses behavioral impairments induced by minimal traumatic brain injury in mice

Social adjustment after closed head injury: a further follow-up seven years after injury

Behavioral consequences of minimal traumatic brain injury in mice.

Enhancing the GLP-1 receptor signaling pathway leads to proliferation and neuroprotection in human neuroblastoma cells.

Apoptosis after traumatic brain injury.

Glutamate receptors, neurotoxicity and neurodegeneration.

Assessment of spatial memory in mice.

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1621-1636

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scholarly article

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10.1007/S11357-012-9464-0

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5

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35

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2012-08-15T00:00:00Z

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22892942

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traumatic brain injury

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3776106

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