Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon. - Wikidata - wikimedia - TriplyDB

Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.

Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.

http://www.wikidata.org/entity/Q37183923

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Novel mitochondrial targets for neuroprotection Identification of εPKC targets during cardiac ischemic injury Mitochondrial import of PKCepsilon is mediated by HSP90: a role in cardioprotection from ischaemia and reperfusion injury HASF is a stem cell paracrine factor that activates PKC epsilon mediated cytoprotection Molecular mechanisms of ischemia-reperfusion injury in brain: pivotal role of the mitochondrial membrane potential in reactive oxygen species generation An effective solution to discover synergistic drugs for anti-cerebral ischemia from traditional Chinese medicinal formulae Essential role of the redox-sensitive kinase p66shc in determining energetic and oxidative status and cell fate in neuronal preconditioning Resveratrol pretreatment protects rat brain from cerebral ischemic damage via a sirtuin 1-uncoupling protein 2 pathway Ischemic preconditioning and clinical scenarios.The neuroprotective mechanism of brain ischemic preconditioning.Protein kinase C epsilon regulates mitochondrial pools of Nampt and NAD following resveratrol and ischemic preconditioning in the rat cortex Ischemic preconditioning mediates cyclooxygenase-2 expression via nuclear factor-kappa B activation in mixed cortical neuronal cultures.Activation of protein kinase C delta following cerebral ischemia leads to release of cytochrome C from the mitochondria via bad pathway.Subanesthetic isoflurane reduces zymosan-induced inflammation in murine Kupffer cells by inhibiting ROS-activated p38 MAPK/NF-κB signaling.Mitochondria: the missing link between preconditioning and neuroprotection.Potential therapeutic benefits of strategies directed to mitochondria.Remote ischemic limb preconditioning after subarachnoid hemorrhage: a phase Ib study of safety and feasibility.Derangements of post-ischemic cerebral blood flow by protein kinase C delta Post-ischemic activation of protein kinase C ε protects the hippocampus from cerebral ischemic injury via alterations in cerebral blood flow.Pathways for ischemic cytoprotection: role of sirtuins in caloric restriction, resveratrol, and ischemic preconditioning.Increased BDNF protein expression after ischemic or PKC epsilon preconditioning promotes electrophysiologic changes that lead to neuroprotection.Epsilon PKC increases brain mitochondrial SIRT1 protein levels via heat shock protein 90 following ischemic preconditioning in rats.Protein kinase C-epsilon activation induces mitochondrial dysfunction and fragmentation in renal proximal tubules Resveratrol Preconditioning Protects Against Cerebral Ischemic Injury via Nuclear Erythroid 2-Related Factor 2.Synaptic mitochondrial pathology in Alzheimer's disease Cardiac mitochondrial matrix and respiratory complex protein phosphorylation Protein Kinase C Epsilon Promotes Cerebral Ischemic Tolerance Via Modulation of Mitochondrial Sirt5 Preconditioning and tolerance against cerebral ischaemia: from experimental strategies to clinical use.New approaches to neuroprotection in infant heart surgery.Global cerebral ischemia: synaptic and cognitive dysfunction.Genetics and genomics of ischemic tolerance: focus on cardiac and cerebral ischemic preconditioning.Oral contraceptives and nicotine synergistically exacerbate cerebral ischemic injury in the female brain.The role of deimination in ATP5b mRNA transport in a transgenic mouse model of multiple sclerosis.C-terminal mechano-growth factor induces heme oxygenase-1-mediated neuroprotection of SH-SY5Y cells via the protein kinase Cϵ/Nrf2 pathway.Ischemia/Reperfusion-Induced Translocation of PKCβII to Mitochondria as an Important Mediator of a Protective Signaling Mechanism in an Ischemia-Resistant Region of the Hippocampus epsilonPKC confers acute tolerance to cerebral ischemic reperfusion injury.Prohibitin reduces mitochondrial free radical production and protects brain cells from different injury modalities.Green tea polyphenols precondition against cell death induced by oxygen-glucose deprivation via stimulation of laminin receptor, generation of reactive oxygen species, and activation of protein kinase Cε.Selective activation of protein kinase C∊ in mitochondria is neuroprotective in vitro and reduces focal ischemic brain injury in mice.Lymphocyte cell kinase activation mediates neuroprotection during ischemic preconditioning.

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Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.

http://www.wikidata.org/entity/Q37183923

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on April 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Ischemic preconditioning targe ...... via protein kinase C epsilon.

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

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JNEUROSCI.5471-07.2008

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Journal of Neuroscience

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Ischemic preconditioning targe ...... via protein kinase C epsilon.

@en

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Alessandra Torraco

http://www.w3.org/2001/XMLSchema#string

Ami P Raval

http://www.w3.org/2001/XMLSchema#string

Antoni Barrientos

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Isabel Saul

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Kunjan R Dave

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Miguel A Perez-Pinzon

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R Anthony DeFazio

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P2860

Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria

The noble gas xenon induces pharmacological preconditioning in the rat heart in vivo via induction of PKC-epsilon and p38 MAPK

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation

The NADH: ubiquinone oxidoreductase (complex I) of the mammalian respiratory chain and the cAMP cascade

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

Mitochondrial PKC epsilon and mitochondrial ATP-sensitive K+ channel copurify and coreconstitute to form a functioning signaling module in proteoliposomes

Identification and localization of an actin-binding motif that is unique to the epsilon isoform of protein kinase C and participates in the regulation of synaptic function

Protein kinase Cepsilon interacts with cytochrome c oxidase subunit IV and enhances cytochrome c oxidase activity in neonatal cardiac myocyte preconditioning

The nitric oxide hypothesis of late preconditioning

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scholarly article

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10.1523/JNEUROSCI.5471-07.2008

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2008-04-01T00:00:00Z

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18417696

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