JNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain. - Wikidata - wikimedia - TriplyDB

JNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain.

JNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain.

http://www.wikidata.org/entity/Q37194288

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Frequency-dependent effects of vibration on peripheral nerves and sensory nerve function in a rat model of hand-arm vibration syndrome. (Erratum: Characterization of frequency-dependent responses of the vascular system Glial cells and chronic pain Resolvins RvE1 and RvD1 attenuate inflammatory pain via central and peripheral actions CXCL5 mediates UVB irradiation-induced pain Interactions of Opioids and HIV Infection in the Pathogenesis of Chronic Pain Mechanisms involved in the development of chemotherapy-induced neuropathy Microglia and spinal cord synaptic plasticity in persistent pain R-flurbiprofen reduces neuropathic pain in rodents by restoring endogenous cannabinoids New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Glial contributions to visceral pain: implications for disease etiology and the female predominance of persistent pain Emerging targets in neuroinflammation-driven chronic pain Soluble epoxide hydrolase expression in a porcine model of arteriovenous graft stenosis and anti-inflammatory effects of a soluble epoxide hydrolase inhibitor NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9 A Wnt5a signaling pathway in the pathogenesis of HIV-1 gp120-induced pain Inhibition of the spinal astrocytic JNK/MCP-1 pathway activation correlates with the analgesic effects of tanshinone IIA sulfonate in neuropathic pain Selective inhibition of JNK with a peptide inhibitor attenuates pain hypersensitivity and tumor growth in a mouse skin cancer pain model Increased chemokine signaling in a model of HIV1-associated peripheral neuropathy.Role of JNK isoforms in the development of neuropathic pain following sciatic nerve transection in the mouse.CCR2 chemokine receptor signaling mediates pain in experimental osteoarthritis.Stat3 inhibition attenuates mechanical allodynia through transcriptional regulation of chemokine expression in spinal astrocytes.Microglial TNFα Induces COX2 and PGI2 Synthase Expression in Spinal Endothelial Cells during Neuropathic Pain.The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition Targeting the Microglial Signaling Pathways: New Insights in the Modulation of Neuropathic Pain.The puerperium alters spinal cord plasticity following peripheral nerve injury.Dynamic effects of TNF-α on synaptic transmission in mice over time following sciatic nerve chronic constriction injury.Expression genetics identifies spinal mechanisms supporting formalin late phase behaviors Chemokine signaling and the management of neuropathic pain.Gp120 in the pathogenesis of human immunodeficiency virus-associated pain.Contribution of the chemokine (C-C motif) ligand 2 (CCL2) to mechanical hypersensitivity after surgical incision in rats.Pathological pain and the neuroimmune interface.Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.Involvement of calcitonin gene-related peptide and CCL2 production in CD40-mediated behavioral hypersensitivity in a model of neuropathic pain Insights into the regulation of chemokine receptors by molecular signaling pathways: functional roles in neuropathic pain Neuronal production of lipocalin-2 as a help-me signal for glial activation Plasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Inhibition of microglial P2X4 receptors attenuates morphine tolerance, Iba1, GFAP and mu opioid receptor protein expression while enhancing perivascular microglial ED2.NADPH oxidase 2-derived reactive oxygen species in spinal cord microglia contribute to peripheral nerve injury-induced neuropathic pain.Tumor necrosis factor alpha sensitizes spinal cord TRPV1 receptors to the endogenous agonist N-oleoyldopamine.Central sensitization: implications for the diagnosis and treatment of pain.A p38 mitogen-activated protein kinase-dependent mechanism of disinhibition in spinal synaptic transmission induced by tumor necrosis factor-alpha.

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JNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain.

http://www.wikidata.org/entity/Q37194288

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article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on April 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

JNK-induced MCP-1 production i ...... tization and neuropathic pain.

@en

JNK-induced MCP-1 production i ...... tization and neuropathic pain.

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JNK-induced MCP-1 production i ...... tization and neuropathic pain.

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JNK-induced MCP-1 production i ...... tization and neuropathic pain.

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JNK-induced MCP-1 production i ...... tization and neuropathic pain.

@en

JNK-induced MCP-1 production i ...... tization and neuropathic pain.

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JNEUROSCI.3623-08.2009

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Journal of Neuroscience

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JNK-induced MCP-1 production i ...... tization and neuropathic pain.

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Anne-Li Lind

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Jong-Yeon Park

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Kawasaki Yasuhiko

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Ling Zhang

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Marc R Suter

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Omar Abdel Samad

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Qiufu Ma

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Zhen-Zhong Xu

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P2860

Chemokines and the pathophysiology of neuropathic pain

Tumor necrosis factor-alpha induces mechanical allodynia after spinal nerve ligation by activation of p38 MAPK in primary sensory neurons

Expression of chemokines and their receptors in human and simian astrocytes: evidence for a central role of TNF alpha and IFN gamma in CXCR4 and CCR5 modulation

Spinal cord repair strategies: why do they work?

Runx1 determines nociceptive sensory neuron phenotype and is required for thermal and neuropathic pain

Regulation of peripheral inflammation by spinal p38 MAP kinase in rats

Impaired neuropathic pain responses in mice lacking the chemokine receptor CCR2

The role of tumor necrosis factor-alpha in the neuropathic pain induced by Lumbar 5 ventral root transection in rat

Excitatory monocyte chemoattractant protein-1 signaling is up-regulated in sensory neurons after chronic compression of the dorsal root ganglion

Metabotropic glutamate receptor subtypes 1 and 5 are activators of extracellular signal-regulated kinase signaling required for inflammatory pain in mice

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4096-4108

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10.1523/JNEUROSCI.3623-08.2009

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13

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2009-04-01T00:00:00Z

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19339605

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neuropathic pain

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2682921

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