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Renal β-intercalated cells maintain body fluid and electrolyte balance

Renal β-intercalated cells maintain body fluid and electrolyte balance

http://www.wikidata.org/entity/Q37200817

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New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptors Molecular mechanisms and regulation of urinary acidification Collecting duct intercalated cell function and regulation Crosstalk between (Pro)renin receptor and COX-2 in the renal medulla during angiotensin II-induced hypertension Prostasin interacts with the epithelial Na+ channel and facilitates cleavage of the γ-subunit by a second protease Pendrin gene ablation alters ENaC subcellular distribution and open probability Deficient acid handling with distal RTA in the NBCe2 knockout mouse.PGE2, Kidney Disease, and Cardiovascular Risk: Beyond Hypertension and Diabetes.An unexpected journey: conceptual evolution of mechanoregulated potassium transport in the distal nephron.Impact of Angiotensin Type 1A Receptors in Principal Cells of the Collecting Duct on Blood Pressure and Hypertension.The role of pendrin in blood pressure regulation.Acetazolamide Attenuates Lithium-Induced Nephrogenic Diabetes Insipidus Resistance to hypertension mediated by intercalated cells of the collecting duct.Electroneutral absorption of NaCl by the aldosterone-sensitive distal nephron: implication for normal electrolytes homeostasis and blood pressure regulation.Chronic kidney disease: targeting prostaglandin E2 receptors.Bicarbonate transport in health and disease.Renal acid-base regulation: new insights from animal models.Relative roles of principal and intercalated cells in the regulation of sodium balance and blood pressure.Activation of mineralocorticoid receptor in salt-sensitive hypertension.Acute regulated expression of pendrin in human urinary exosomes.Context-dependent mechanisms modulating aldosterone signaling in the kidney.Potassium: friend or foe?Glucagon actions on the kidney revisited: possible role in potassium homeostasis.Acidosis and Urinary Calcium Excretion: Insights from Genetic Disorders.Effect of diuretics on renal tubular transport of calcium and magnesium.Uroguanylin inhibits H-ATPase activity and surface expression in renal distal tubules by a PKG-dependent pathway Opening lines of communication in the distal nephron.Integrated compensatory network is activated in the absence of NCC phosphorylation Molecular architecture underlying fluid absorption by the developing inner ear Renal Atp6ap2/(Pro)renin Receptor Is Required for Normal Vacuolar H+-ATPase Function but Not for the Renin-Angiotensin System.Basic research: Salt wasting in distal renal tubular acidosis-new look, old problem.Adaptive changes in GFR, tubular morphology, and transport in subtotal nephrectomized kidneys: modeling and analysis.Renal intercalated cells and blood pressure regulation.The ClC-K2 Chloride Channel Is Critical for Salt Handling in the Distal Nephron.Lithium increases ammonium excretion leading to altered urinary acid-base buffer composition.New Findings on the Pathogenesis of Distal Renal Tubular Acidosis.Aldosterone Is Essential for Angiotensin II-Induced Upregulation of Pendrin.Versatility of NaCl transport mechanisms in the cortical collecting duct.Pathophysiology, diagnosis and treatment of inherited distal renal tubular acidosis.The mechanosensitive BKα/β1 channel localizes to cilia of principal cells in rabbit cortical collecting duct (CCD).

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Renal β-intercalated cells maintain body fluid and electrolyte balance

http://www.wikidata.org/entity/Q37200817

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 24 September 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Renal β-intercalated cells maintain body fluid and electrolyte balance

@en

Renal β-intercalated cells maintain body fluid and electrolyte balance.

@nl

type

label

Renal β-intercalated cells maintain body fluid and electrolyte balance

@en

Renal β-intercalated cells maintain body fluid and electrolyte balance.

@nl

prefLabel

Renal β-intercalated cells maintain body fluid and electrolyte balance

@en

Renal β-intercalated cells maintain body fluid and electrolyte balance.

@nl

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Journal of Clinical Investigation

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Renal β-intercalated cells maintain body fluid and electrolyte balance

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P2093

Carsten A Wagner

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Fabien Sohet

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Françoise Leviel

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Marion Vallet

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Maximilien Jayat

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Régine Chambrey

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Victor Gueutin

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P2860

Mutations in the gene encoding B1 subunit of H+-ATPase cause renal tubular acidosis with sensorineural deafness

Mutations in ATP6N1B, encoding a new kidney vacuolar proton pump 116-kD subunit, cause recessive distal renal tubular acidosis with preserved hearing

Molecular cloning and characterization of Atp6v1b1, the murine vacuolar H+ -ATPase B1-subunit

Localization and regulation of the ATP6V0A4 (a4) vacuolar H+-ATPase subunit defective in an inherited form of distal renal tubular acidosis

Familial distal renal tubular acidosis is associated with mutations in the red cell anion exchanger (Band 3, AE1) gene

Pendrin, encoded by the Pendred syndrome gene, resides in the apical region of renal intercalated cells and mediates bicarbonate secretion

Mutations in the chloride-bicarbonate exchanger gene AE1 cause autosomal dominant but not autosomal recessive distal renal tubular acidosis

Genetic diseases of acid-base transporters

Identification of a chloride-formate exchanger expressed on the brush border membrane of renal proximal tubule cells

The B1-subunit of the H(+) ATPase is required for maximal urinary acidification

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4219-4231

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scholarly article

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10.1172/JCI63492

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10

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123

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Janos Peti-Peterdi

Dominique Eladari

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2013-09-24T00:00:00Z

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24051376

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3784520

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