Integrated compensatory network is activated in the absence of NCC phosphorylation
about
New insights into sodium transport regulation in the distal nephron: Role of G-protein coupled receptorsEmerging Targets of Diuretic Therapy.Transcription factor TFCP2L1 patterns cells in the mouse kidney collecting ducts.Systems biology of diuretic resistance.Effect of NBCe1 deletion on renal citrate and 2-oxoglutarate handling.The role of pendrin in blood pressure regulation.The CUL3/KLHL3-WNK-SPAK/OSR1 pathway as a target for antihypertensive therapyWNK signalling pathways in blood pressure regulationUrinary proteomics for the study of genetic kidney diseases.Potassium: friend or foe?Unsung renal receptors: orphan G-protein-coupled receptors play essential roles in renal development and homeostasis.SPAK and OSR1 play essential roles in potassium homeostasis through actions on the distal convoluted tubule.Direct and Indirect Mineralocorticoid Effects Determine Distal Salt Transport.Single-tubule RNA-Seq uncovers signaling mechanisms that defend against hyponatremia in SIADH.Dietary sodium induces a redistribution of the tubular metabolic workload.Identifying the localization and exploring a functional role for Gprc5c in the kidney.Renal and colonic potassium transporters in the pregnant rat.Diuretic Treatment in Heart Failure.Expression of sodium-dependent dicarboxylate transporter 1 (NaDC1/SLC13A2) in normal and neoplastic human kidney.Consequences of SPAK inactivation on Hyperkalemic Hypertension caused by WNK1 mutations: evidence for differential roles of WNK1 and WNK4.Double Knockout of the Na+-Driven Cl-/HCO3- Exchanger and Na+/Cl- Cotransporter Induces Hypokalemia and Volume Depletion.α-ketoglutarate stimulates pendrin-dependent Cl- absorption in the mouse CCD through protein kinase C.Renal Tubular Ubiquitin-Protein Ligase NEDD4-2 Is Required for Renal Adaptation during Long-Term Potassium Depletion.Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules.The complexity of diuretic resistance.Diuretic Resistance.
P2860
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P2860
Integrated compensatory network is activated in the absence of NCC phosphorylation
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Integrated compensatory network is activated in the absence of NCC phosphorylation
@en
Integrated compensatory network is activated in the absence of NCC phosphorylation
@nl
type
label
Integrated compensatory network is activated in the absence of NCC phosphorylation
@en
Integrated compensatory network is activated in the absence of NCC phosphorylation
@nl
prefLabel
Integrated compensatory network is activated in the absence of NCC phosphorylation
@en
Integrated compensatory network is activated in the absence of NCC phosphorylation
@nl
P2093
P2860
P356
P1476
Integrated compensatory network is activated in the absence of NCC phosphorylation
@en
P2093
Edward J Weinman
Eric Delpire
James B Wade
Paul A Welling
Richard Coleman
Susan G Dorsey
Susan M Wall
P2860
P304
P356
10.1172/JCI78558
P407
P577
2015-04-20T00:00:00Z