Suppression of native defense mechanisms, SIRT1 and PPARγ, by dietary glycoxidants precedes disease in adult humans; relevance to lifestyle-engendered chronic diseases.

Suppression of native defense mechanisms, SIRT1 and PPARγ, by dietary glycoxidants precedes disease in adult humans; relevance to lifestyle-engendered chronic diseases.

Item
http://www.wikidata.org/entity/Q37227877
Dietary Advanced Glycation End Products and Risk Factors for Chronic Disease: A Systematic Review of Randomised Controlled TrialsAdvanced glycation end-products: modifiable environmental factors profoundly mediate insulin resistanceOral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humansConsumption of diets with low advanced glycation end products improves cardiometabolic parameters: meta-analysis of randomised controlled trials.Effect of dietary advanced glycation end products on inflammation and cardiovascular risks in healthy overweight adults: a randomised crossover trialDietary fat quantity and quality modifies advanced glycation end products metabolism in patients with metabolic syndrome.Sirt1 deletion leads to enhanced inflammation and aggravates endotoxin-induced acute kidney injuryElevated serum advanced glycation endproducts in obese indicate risk for the metabolic syndrome: a link between healthy and unhealthy obesity?The Possible Mechanism of Advanced Glycation End Products (AGEs) for Alzheimer's DiseaseInhibition of Advanced Glycation End Products (AGEs) Accumulation by Pyridoxamine Modulates Glomerular and Mesangial Cell Estrogen Receptor α Expression in Aged Female Mice.Effect of diet-derived advanced glycation end products on inflammation.Dietary Advanced Glycation End Products and Cardiometabolic Risk.Oral AGE restriction ameliorates insulin resistance in obese individuals with the metabolic syndrome: a randomised controlled trial.A Retrospective Study in Adults with Metabolic Syndrome: Diabetic Risk Factor Response to Daily Consumption of Agaricus bisporus (White Button Mushrooms).Dietary advanced glycated end-products and medicines influence the expression of SIRT1 and DDOST in peripheral mononuclear cells from long-term type 1 diabetes patients.Mediterranean Diet Supplemented With Coenzyme Q10 Modulates the Postprandial Metabolism of Advanced Glycation End Products in Elderly Men and Women.
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Suppression of native defense mechanisms, SIRT1 and PPARγ, by dietary glycoxidants precedes disease in adult humans; relevance to lifestyle-engendered chronic diseases.

Item
http://www.wikidata.org/entity/Q37227877
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Suppression of native defense ...... e-engendered chronic diseases.
@en
Suppression of native defense ...... e-engendered chronic diseases.
@nl
type
label
Suppression of native defense ...... e-engendered chronic diseases.
@en
Suppression of native defense ...... e-engendered chronic diseases.
@nl
prefLabel
Suppression of native defense ...... e-engendered chronic diseases.
@en
Suppression of native defense ...... e-engendered chronic diseases.
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
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P1433
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Suppression of native defense ...... e-engendered chronic diseases.
@en
P2093
P2860
P2888
P304
P31
P356
P433
P478
P577
P698
P932