Sulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment. - Wikidata - wikimedia - TriplyDB

Sulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment.

Sulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment.

http://www.wikidata.org/entity/Q37232070

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The Role of the Reactive Oxygen Species and Oxidative Stress in the Pathomechanism of the Age-Related Ocular Diseases and Other Pathologies of the Anterior and Posterior Eye Segments in Adults Decreased DJ-1 leads to impaired Nrf2-regulated antioxidant defense and increased UV-A-induced apoptosis in corneal endothelial cells.UV-A Irradiation Activates Nrf2-Regulated Antioxidant Defense and Induces p53/Caspase3-Dependent Apoptosis in Corneal Endothelial Cells.Activation of mitophagy leads to decline in Mfn2 and loss of mitochondrial mass in Fuchs endothelial corneal dystrophy.Sulforaphane Induced Apoptosis via Promotion of Mitochondrial Fusion and ERK1/2-Mediated 26S Proteasome Degradation of Novel Pro-survival Bim and Upregulation of Bax in Human Non-Small Cell Lung Cancer Cells Association between corneal endothelial cell densities and elevated cytokine levels in the aqueous humor.Nrf2, a Potential Therapeutic Target against Oxidative Stress in Corneal Diseases.Hanging drop monoculture for selection of optimal antioxidants during in vitro maturation of porcine oocytes.NQO1 downregulation potentiates menadione-induced endothelial-mesenchymal transition during rosette formation in Fuchs endothelial corneal dystrophy.DJ-1 in Ocular Diseases: A Review.Perspective of Future Potent Therapies for Fuchs Endothelial Corneal Dystrophy Sulforaphane protects rabbit corneas against oxidative stress injury in keratoconus through activation of the Nrf-2/HO-1 antioxidant pathway

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Sulforaphane decreases endothelial cell apoptosis in fuchs endothelial corneal dystrophy: a novel treatment.

http://www.wikidata.org/entity/Q37232070

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 15 October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Sulforaphane decreases endothe ...... dystrophy: a novel treatment.

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Investigative Ophthalmology & Visual Science

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Sulforaphane decreases endothe ...... l dystrophy: a novel treatment

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Alireza Ziaei

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Thore Schmedt

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Yuming Chen

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P2860

Cruciferous vegetables and human cancer risk: epidemiologic evidence and mechanistic basis

Molecular targets of dietary phenethyl isothiocyanate and sulforaphane for cancer chemoprevention

Identification of Nrf2-regulated genes induced by the chemopreventive agent sulforaphane by oligonucleotide microarray

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression

Stabilization of Nrf2 by tBHQ confers protection against oxidative stress-induced cell death in human neural stem cells.

NRF2 activation restores disease related metabolic deficiencies in olfactory neurosphere-derived cells from patients with sporadic Parkinson's disease

Evidence of oxidative stress in the pathogenesis of fuchs endothelial corneal dystrophy.

Potency of Michael reaction acceptors as inducers of enzymes that protect against carcinogenesis depends on their reactivity with sulfhydryl groups

Multi-targeted prevention of cancer by sulforaphane.

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6724-6734

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scholarly article

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10.1167/IOVS.13-12699

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apoptotic process

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3797593

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