Impaired insulin exocytosis in neural cell adhesion molecule-/- mice due to defective reorganization of the submembrane F-actin network
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N-cadherin is dispensable for pancreas development but required for beta-cell granule turnover.Insulin-producing cells from human adipose tissue-derived mesenchymal stem cells detected by atomic force microscope.Phosphatidylinositol-4-phosphate-5-kinase alpha deficiency alters dynamics of glucose-stimulated insulin release to improve glucohomeostasis and decrease obesity in mice.Insulin-producing cells could not mimic the physiological regulation of insulin secretion performed by pancreatic beta cells.N-cadherin loss in POMC-expressing cells leads to pituitary disorganizationThe pancreatic beta cell surface proteome.EphA4 Receptor Forward Signaling Inhibits Glucagon Secretion From α-Cells.Kin of IRRE-like Protein 2 Is a Phosphorylated Glycoprotein That Regulates Basal Insulin Secretion.PPARβ/δ affects pancreatic β cell mass and insulin secretion in mice.A p21-activated kinase (PAK1) signaling cascade coordinately regulates F-actin remodeling and insulin granule exocytosis in pancreatic β cells.Sympathetic innervation during development is necessary for pancreatic islet architecture and functional maturation.Insulin-regulated glucagon-like peptide-1 release from L cells: actin' out.Islet organogenesis, angiogenesis and innervation.Effect of Cell Adhesion Molecule 1 Expression on Intracellular Granule Movement in Pancreatic α Cells.The triggering pathway to insulin secretion: Functional similarities and differences between the human and the mouse β cells and their translational relevance.Gap junctions and other mechanisms of cell-cell communication regulate basal insulin secretion in the pancreatic islet.PKA-independent cAMP stimulation of white adipocyte exocytosis and adipokine secretion: modulations by Ca2+ and ATP.Cellular modeling: insight into oral minimal models of insulin secretion.Pancreatic β-Cell Electrical Activity and Insulin Secretion: Of Mice and Men.Signals in the pancreatic islet microenvironment influence β-cell proliferation.
P2860
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P2860
Impaired insulin exocytosis in neural cell adhesion molecule-/- mice due to defective reorganization of the submembrane F-actin network
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 12 February 2009
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Impaired insulin exocytosis in ...... he submembrane F-actin network
@en
Impaired insulin exocytosis in ...... e submembrane F-actin network.
@nl
type
label
Impaired insulin exocytosis in ...... he submembrane F-actin network
@en
Impaired insulin exocytosis in ...... e submembrane F-actin network.
@nl
prefLabel
Impaired insulin exocytosis in ...... he submembrane F-actin network
@en
Impaired insulin exocytosis in ...... e submembrane F-actin network.
@nl
P2093
P2860
P50
P356
P1433
P1476
Impaired insulin exocytosis in ...... he submembrane F-actin network
@en
P2093
Charlotta S Olofsson
Chris Partridge
Ingmar Lundquist
Joakim Håkansson
Juris Galvanovskis
Maria SörhedeWinzell
Martin Bengtsson
Xiaojie Xian
P2860
P304
P356
10.1210/EN.2008-0475
P407
P577
2009-02-12T00:00:00Z