Homing of GAD65 specific autoimmunity and development of insulitis requires expression of both DQ8 and human GAD65 in transgenic mice.
about
Pearls in autoimmunityEukaryotic translation initiation factor 5A inhibition alters physiopathology and immune responses in a "humanized" transgenic mouse model of type 1 diabetes.Glucose intolerance and diabetes following antigen-specific insulitis in diabetes-susceptible "humanized" transgenic mice.Diabetes-associated dry eye syndrome in a new humanized transgenic model of type 1 diabetes.
P2860
Homing of GAD65 specific autoimmunity and development of insulitis requires expression of both DQ8 and human GAD65 in transgenic mice.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 16 March 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@en
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@nl
type
label
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@en
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@nl
prefLabel
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@en
Homing of GAD65 specific autoi ...... uman GAD65 in transgenic mice.
@nl
P2093
P2860
P1476
Homing of GAD65 specific autoi ...... human GAD65 in transgenic mice
@en
P2093
Maria J Diacovo
Raya B Elagin
Sadguna Balijepalli
P2860
P356
10.1016/J.JAUT.2009.02.004
P577
2009-03-16T00:00:00Z