Molecular correlates of altered expression of potassium currents in failing rabbit myocardium.
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Ion Channels in the HeartCaMKII regulation of cardiac K channelsElectrophysiology of Heart Failure Using a Rabbit Model: From the Failing Myocyte to Ventricular Fibrillation.Voltage-gated ion channel Kv4.3 is associated with Rap guanine nucleotide exchange factors and regulates angiotensin receptor type 1 signaling to small G-protein RapLong-term fish oil supplementation induces cardiac electrical remodeling by changing channel protein expression in the rabbit model.Electrical remodeling in the failing heart.Increased short-term variability of the QT interval in professional soccer players: possible implications for arrhythmia prediction.IKs protects from ventricular arrhythmia during cardiac ischemia and reperfusion in rabbits by preserving the repolarization reserve.Cdon deficiency causes cardiac remodeling through hyperactivation of WNT/β-catenin signalingEnhanced cardiac PI3Kα signalling mitigates arrhythmogenic electrical remodelling in pathological hypertrophy and heart failureRepolarization reserve evolves dynamically during the cardiac action potential: effects of transient outward currents on early afterdepolarizations.Mechanisms of ventricular arrhythmias: a dynamical systems-based perspective.Simulation of Ca-calmodulin-dependent protein kinase II on rabbit ventricular myocyte ion currents and action potentials.Basic science of cardiac resynchronization therapy: molecular and electrophysiological mechanismsRGS2 overexpression or G(i) inhibition rescues the impaired PKA signaling and slow AP firing of cultured adult rabbit pacemaker cellsBradycardia-mediated ventricular electrical remodeling.Regulation of the instantaneous inward rectifier and the delayed outward rectifier potassium channels by Captopril and Angiotensin II via the Phosphoinositide-3 kinase pathway in volume-overload-induced hypertrophied cardiac myocytes.Prevention of adverse electrical and mechanical remodeling with biventricular pacing in a rabbit model of myocardial infarction.Heart failure duration progressively modulates the arrhythmia substrate through structural and electrical remodeling.Bisoprolol reverses down-regulation of potassium channel proteins in ventricular tissues of rabbits with heart failure.Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation.Modulation of ventricular transient outward K⁺ current by acidosis and its effects on excitation-contraction coupling.Distinct cellular and molecular mechanisms underlie functional remodeling of repolarizing K+ currents with left ventricular hypertrophy.Electrophysiological consequences of dyssynchronous heart failure and its restoration by resynchronization therapy.Remodelling of cardiac repolarization: how homeostatic responses can lead to arrhythmogenesis.Electrical remodeling contributes to complex tachyarrhythmias in connexin43-deficient mouse hearts.Revisiting K(+) channel-dependent electrical remodeling in the border zone.SAP97 regulates Kir2.3 channels by multiple mechanisms.Genomics, transcriptional profiling, and heart failure.Unfolded protein response regulates cardiac sodium current in systolic human heart failureElectrical remodeling in dyssynchrony and resynchronization.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.Arrhythmogenic and metabolic remodelling of failing human heart.Transmural gradients in ion channel and auxiliary subunit expression.Effect of right ventricular versus biventricular pacing on electrical remodeling in the normal heart.Inward rectifier potassium channels in the HL-1 cardiomyocyte-derived cell line.A novel dual-fluorescence strategy for functionally validating microRNA targets in 3' untranslated regions: regulation of the inward rectifier potassium channel K(ir)2.1 by miR-212.Arrhythmogenic remodelling of activation and repolarization in the failing human heart.Mitogen-activated protein kinases control cardiac KChIP2 gene expression.Differential responses of rabbit ventricular and atrial transient outward current (Ito) to the Ito modulator NS5806.
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Molecular correlates of altered expression of potassium currents in failing rabbit myocardium.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 January 2005
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Molecular correlates of altere ...... in failing rabbit myocardium.
@en
Molecular correlates of altere ...... in failing rabbit myocardium.
@nl
type
label
Molecular correlates of altere ...... in failing rabbit myocardium.
@en
Molecular correlates of altere ...... in failing rabbit myocardium.
@nl
prefLabel
Molecular correlates of altere ...... in failing rabbit myocardium.
@en
Molecular correlates of altere ...... in failing rabbit myocardium.
@nl
P2093
P2860
P1476
Molecular correlates of altere ...... s in failing rabbit myocardium
@en
P2093
Antonis A Armoundas
Brian O'Rourke
David A Kass
Deborah DiSilvestre
Eduardo Marbán
Gordon F Tomaselli
Jochen Rose
Miroslava Burysek
Yanli Tian
P2860
P304
P356
10.1152/AJPHEART.00526.2003
P577
2005-01-06T00:00:00Z