Oncogenic conversion of transcription factors by chromosomal translocations.
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Cloning and characterization of the EAP30 subunit of the ELL complex that confers derepression of transcription by RNA polymerase IIHepatitis C virus core protein-induced loss of LZIP function correlates with cellular transformationStructural basis for the heterodimeric interaction between the acute leukaemia-associated transcription factors AML1 and CBFbetaFusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcomaInteraction of p53 with the human Rad51 proteinThe PAX3-FKHR fusion protein created by the t(2;13) translocation in alveolar rhabdomyosarcomas is a more potent transcriptional activator than PAX3Structure, localization and transcriptional properties of two classes of retinoic acid receptor alpha fusion proteins in acute promyelocytic leukemia (APL): structural similarities with a new family of oncoproteinsThe LIM protein RBTN2 and the basic helix-loop-helix protein TAL1 are present in a complex in erythroid cellsThe t(8;21) translocation in acute myeloid leukemia results in production of an AML1-MTG8 fusion transcriptFusion between a novel Krüppel-like zinc finger gene and the retinoic acid receptor-alpha locus due to a variant t(11;17) translocation associated with acute promyelocytic leukaemiaGeneration of the AML1-EVI-1 fusion gene in the t(3;21)(q26;q22) causes blastic crisis in chronic myelocytic leukemiaDNA-binding and transcriptional regulatory properties of hepatic leukemia factor (HLF) and the t(17;19) acute lymphoblastic leukemia chimera E2A-HLFDNA-binding specificity and trans-activating potential of the leukemia-associated E2A-hepatic leukemia factor fusion proteinHOX11 interacts with CTF1 and mediates hematopoietic precursor cell immortalizationA novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOPThymic overexpression of Ttg-1 in transgenic mice results in T-cell acute lymphoblastic leukemia/lymphomaMouse Af9 is a controller of embryo patterning, like Mll, whose human homologue fuses with Af9 after chromosomal translocation in leukemiaThe molecular basis of genetic dominanceENL, the MLL fusion partner in t(11;19), binds to the c-Abl interactor protein 1 (ABI1) that is fused to MLL in t(10;11)+.Disrupted differentiation and oncogenic transformation of lymphoid progenitors in E2A-HLF transgenic miceChildhood acute lymphoblastic leukemia: is there a tumor suppressor gene in chromosome 12p12.3?Immortalization and leukemic transformation of a myelomonocytic precursor by retrovirally transduced HRX-ENL.The t(9;14)(p13;q32) translocation in B-cell non-Hodgkin's lymphoma.The oncogenic capacity of HRX-ENL requires the transcriptional transactivation activity of ENL and the DNA binding motifs of HRX.Rearrangement of the PAX3 paired box gene in the paediatric solid tumour alveolar rhabdomyosarcoma.Molecular mechanisms of leukemogenesis mediated by MLL fusion proteins.Identification of SAS4 and SAS5, two genes that regulate silencing in Saccharomyces cerevisiae.Cytogenetic abnormalities in childhood acute lymphoblastic leukemia correlates with clinical features and treatment outcome.Transcriptional activation by TAL1 and FUS-CHOP proteins expressed in acute malignancies as a result of chromosomal abnormalities.Conditional Expression of E2A-HLF Induces B-Cell Precursor Death and Myeloproliferative-Like Disease in Knock-In Mice.Growth inhibition and induction of differentiation of t(8;21) acute myeloid leukemia cells by the DNA-binding domain of PEBP2 and the AML1/MTG8(ETO)-specific antisense oligonucleotide.Growth stimulation of primary B cell precursors by the anti-phosphatase Sbf1.Analysis of the murine All-1 gene reveals conserved domains with human ALL-1 and identifies a motif shared with DNA methyltransferases.Homologous recombination repair protects against particulate chromate-induced chromosome instability in Chinese hamster cells.Dual functions of the AML1/Evi-1 chimeric protein in the mechanism of leukemogenesis in t(3;21) leukemias.Preferential repair of UV damage in highly transcribed DNA diminishes UV-induced intrachromosomal recombination in mammalian cellsMechanisms of transcription factor deregulation in lymphoid cell transformation.Transformation by homeobox genes can be mediated by selective transcriptional repression.A mutant v-rel with increased ability to transform B lymphocytes.LMO2 at 25 years: a paradigm of chromosomal translocation proteins.
P2860
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P2860
Oncogenic conversion of transcription factors by chromosomal translocations.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on August 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Oncogenic conversion of transcription factors by chromosomal translocations.
@en
Oncogenic conversion of transcription factors by chromosomal translocations.
@nl
type
label
Oncogenic conversion of transcription factors by chromosomal translocations.
@en
Oncogenic conversion of transcription factors by chromosomal translocations.
@nl
prefLabel
Oncogenic conversion of transcription factors by chromosomal translocations.
@en
Oncogenic conversion of transcription factors by chromosomal translocations.
@nl
P1433
P1476
Oncogenic conversion of transcription factors by chromosomal translocations.
@en
P2093
P304
P356
10.1016/0092-8674(91)90105-8
P407
P577
1991-08-01T00:00:00Z