The model homologue of the partially defective human 5,10-methylenetetrahydrofolate reductase, considered as a risk factor for stroke due to increased homocysteine level, can be protected and reactivated by heat shock proteins.
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The model homologue of the partially defective human 5,10-methylenetetrahydrofolate reductase, considered as a risk factor for stroke due to increased homocysteine level, can be protected and reactivated by heat shock proteins.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 May 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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The model homologue of the par ...... ivated by heat shock proteins.
@en
The model homologue of the par ...... ivated by heat shock proteins.
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type
label
The model homologue of the par ...... ivated by heat shock proteins.
@en
The model homologue of the par ...... ivated by heat shock proteins.
@nl
prefLabel
The model homologue of the par ...... ivated by heat shock proteins.
@en
The model homologue of the par ...... ivated by heat shock proteins.
@nl
P2860
P50
P1476
The model homologue of the par ...... ivated by heat shock proteins.
@en
P2093
Leszek Kadziński
Michał Grabowski
P2860
P2888
P304
P356
10.1007/S11011-016-9844-8
P50
P577
2016-05-28T00:00:00Z