Effects of flavonoids on glycosaminoglycan synthesis: implications for substrate reduction therapy in Sanfilippo disease and other mucopolysaccharidoses.The model homologue of the partially defective human 5,10-methylenetetrahydrofolate reductase, considered as a risk factor for stroke due to increased homocysteine level, can be protected and reactivated by heat shock proteins.Factors and processes modulating phenotypes in neuronopathic lysosomal storage diseases.Putative biological mechanisms of efficiency of substrate reduction therapies for mucopolysaccharidoses.Genistein inhibits activities of methylenetetrahydrofolate reductase and lactate dehydrogenase, enzymes which use NADH as a substrate.Increased levels of antibodies against heat shock proteins in stroke patients.[Mineralization of the basal ganglia as the supposed cause of poor tolerance of zuclopenthixol in a patient with long-term untreated paranoid schizophrenia].Glycosaminoglycans and mucopolysaccharidosis type III.Homocysteine level and metabolism in ischemic stroke in the population of Northern Poland.Improved HPLC method for total plasma homocysteine detection and quantification.Evidence for interactions between homocysteine and genistein: insights into stroke risk and potential treatment.[Anticardiolopin antibodies as a risk factor in stroke][New diagnostic criteria for the neuropsychiatric form of systemic lupus erythematosus][Evaluation of long term results of surgery for myasthenia gravis (with regard to electrophysiological studies)]
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1950-01-01T00:00:00Z
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