Airway hyperresponsiveness in the absence of CD4+ T cells after primary but not secondary challenge
about
Understanding asthma using animal modelsOvalbumin sensitization of guinea pig at birth prevents the ontogenetic decrease in airway smooth muscle responsiveness.Microbial heat shock protein 65 attenuates airway hyperresponsiveness and inflammation by modulating the function of dendritic cells.Differential effects of dendritic cell transfer on airway hyperresponsiveness and inflammation.Spectrum of T-lymphocyte activities regulating allergic lung inflammation.Attenuation of allergen-induced airway hyperresponsiveness is mediated by airway regulatory T cells.Natural killer T cells regulate the development of asthma.
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Airway hyperresponsiveness in the absence of CD4+ T cells after primary but not secondary challenge
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 21 April 2005
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Airway hyperresponsiveness in ...... ry but not secondary challenge
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Airway hyperresponsiveness in ...... y but not secondary challenge.
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type
label
Airway hyperresponsiveness in ...... ry but not secondary challenge
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Airway hyperresponsiveness in ...... y but not secondary challenge.
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prefLabel
Airway hyperresponsiveness in ...... ry but not secondary challenge
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Airway hyperresponsiveness in ...... y but not secondary challenge.
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P2860
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P1476
Airway hyperresponsiveness in ...... ry but not secondary challenge
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Anthony Joetham
Arihiko Kanehiro
Christian Taube
Katsuyuki Takeda
Nobuaki Miyahara
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P356
10.1165/RCMB.2004-0414OC
P577
2005-04-21T00:00:00Z