MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers. - Wikidata - wikimedia - TriplyDB

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

http://www.wikidata.org/entity/Q37274389

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mRNAs and miRNAs in whole blood associated with lung hyperplasia, fibrosis, and bronchiolo-alveolar adenoma and adenocarcinoma after multi-walled carbon nanotube inhalation exposure in mice Increased miR-708 expression in NSCLC and its association with poor survival in lung adenocarcinoma from never smokers Overlapping functions of microRNAs in control of apoptosis during Drosophila embryogenesis Molecular biology of lung cancer: clinical implications Rho-associated kinases in tumorigenesis: re-considering ROCK inhibition for cancer therapy MicroRNA In Lung Cancer: Novel Biomarkers and Potential Tools for Treatment Role of microRNAs in chemoresistance MicroRNAs and Growth Factors: An Alliance Propelling Tumor Progression Emerging Roles of MicroRNAs in EGFR-Targeted Therapies for Lung Cancer MicroRNAs in respiratory disease. A clinician's overview SNPing cancer in the bud: microRNA and microRNA-target site polymorphisms as diagnostic and prognostic biomarkers in cancer Non-coding RNAs in lung cancer Alterations of microRNAs contribute to colon carcinogenesis Can microRNAs improve the management of lung cancer patients? A clinician's perspective Role of Exosomal Noncoding RNAs in Lung Carcinogenesis MicroRNAs in flow-dependent vascular remodelling Regulating miRNA by natural agents as a new strategy for cancer treatment Enzastaurin has anti-tumour effects in lung cancers with overexpressed JAK pathway molecules.MicroRNA-21 modulates the levels of reactive oxygen species by targeting SOD3 and TNFα Suppression of miR-204 enables oral squamous cell carcinomas to promote cancer stemness, EMT traits, and lymph node metastasis The role of epigenetics in resistance to Cisplatin chemotherapy in lung cancer Overexpression of deubiquitinating enzyme USP28 promoted non-small cell lung cancer growth Bioinformatics of Cancer ncRNA in High Throughput Sequencing: Present State and Challenges Inflammation and cancer: interweaving microRNA, free radical, cytokine and p53 pathways.Non-small cell lung cancer associated microRNA expression signature: integrated bioinformatics analysis, validation and clinical significance.Cigarette smoke induces C/EBP-β-mediated activation of miR-31 in normal human respiratory epithelia and lung cancer cells.ABCG1 as a potential oncogene in lung cancer The new concepts on overcoming drug resistance in lung cancer.MicroRNA Signature of Lung Adenocarcinoma with EGFR Exon 19 Deletion Dehydroepiandrosterone-induces miR-21 transcription in HepG2 cells through estrogen receptor β and androgen receptor Molecular mechanisms of acquired resistance to tyrosine kinase targeted therapy The low expression of miR-451 predicts a worse prognosis in non-small cell lung cancer cases.Implication of microRNAs in drug resistance for designing novel cancer therapy microRNAs and lung cancer: tumors and 22-mers.MicroRNA expression distinguishes SCLC from NSCLC lung tumor cells and suggests a possible pathological relationship between SCLCs and NSCLCs miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis Identification of a microRNA signature of renal ischemia reperfusion injury.MicroRNAs and lung cancer: Biology and applications in diagnosis and prognosis.MicroRNA-21 is involved in ionizing radiation-promoted liver carcinogenesis.Downregulation of the Rho GTPase signaling pathway is involved in the microRNA-138-mediated inhibition of cell migration and invasion in tongue squamous cell carcinoma.

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MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

http://www.wikidata.org/entity/Q37274389

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 13 July 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@en

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@nl

type

label

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@en

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@nl

prefLabel

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@en

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

@nl

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PNAS.0905234106

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Proceedings of the National Academy of Sciences of the United States of America

P1476

MiR-21 is an EGFR-regulated anti-apoptotic factor in lung cancer in never-smokers.

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Aaron J Schetter

http://www.w3.org/2001/XMLSchema#string

Akihiko Gemma

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Akiteru Goto

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Curtis C Harris

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Elise D Bowman

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Ewy A Mathe

http://www.w3.org/2001/XMLSchema#string

Haruhiko Sugimura

http://www.w3.org/2001/XMLSchema#string

Izumi Horikawa

http://www.w3.org/2001/XMLSchema#string

Jin Jen

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Masahiro Seike

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P2860

miR-21 Gene expression triggered by AP-1 is sustained through a double-negative feedback mechanism

Frequent deletions and down-regulation of micro- RNA genes miR15 and miR16 at 13q14 in chronic lymphocytic leukemia

A microRNA expression signature of human solid tumors defines cancer gene targets

Somatic mutations affect key pathways in lung adenocarcinoma

MicroRNA expression profiles associated with prognosis and therapeutic outcome in colon adenocarcinoma

EGFR mutations in lung cancer: correlation with clinical response to gefitinib therapy.

EGFR mutation and resistance of non-small-cell lung cancer to gefitinib.

Allelic dilution obscures detection of a biologically significant resistance mutation in EGFR-amplified lung cancer.

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling.

RAS is regulated by the let-7 microRNA family

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12085-12090

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scholarly article

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10.1073/PNAS.0905234106

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29

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106

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Carlo Maria Croce

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2009-07-13T00:00:00Z

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26665836

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19597153

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apoptotic process

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2715493

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