Smooth muscle cells give rise to osteochondrogenic precursors and chondrocytes in calcifying arteries. - Wikidata - wikimedia - TriplyDB

Smooth muscle cells give rise to osteochondrogenic precursors and chondrocytes in calcifying arteries.

Smooth muscle cells give rise to osteochondrogenic precursors and chondrocytes in calcifying arteries.

http://www.wikidata.org/entity/Q37276028

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A role for the endothelium in vascular calcification Large animal models of cardiovascular disease Diabetic Cardiovascular Disease Induced by Oxidative Stress Animal Models to Study Links between Cardiovascular Disease and Renal Failure and Their Relevance to Human Pathology Regulatory circuits controlling vascular cell calcification Arterial calcification in chronic kidney disease: key roles for calcium and phosphate Markers for the identification of tendon-derived stem cells in vitro and tendon stem cells in situ - update and future development Cardiovascular Risk Factors and Chronic Kidney Disease-FGF23: A Key Molecule in the Cardiovascular Disease Vascular calcifying progenitor cells possess bidirectional differentiation potentials Smooth muscle cell plasticity: fact or fiction?Smooth muscle cell phenotypic switching in atherosclerosis Vascular smooth muscle cells in cerebral aneurysm pathogenesis.Matrix Gla protein deficiency causes arteriovenous malformations in mice Association of calcium concentration with pulse pressure in older women: data from a large population-based multicentric study.Arterial Stiffness: A Nexus between Cardiac and Renal Disease.Calcification in dermal fibroblasts from a patient with GGCX syndrome accompanied by upregulation of osteogenic molecules Systems biology of vascular calcification Mesenchymal stem cells recruited by active TGFβ contribute to osteogenic vascular calcification Regulation of vascular smooth muscle cell calcification by extracellular pyrophosphate homeostasis: synergistic modulation by cyclic AMP and hyperphosphatemia A smooth muscle-like origin for beige adipocytes Inflammation and the osteogenic regulation of vascular calcification: a review and perspective.Apelin attenuates the osteoblastic differentiation of vascular smooth muscle cells.Has our understanding of calcification in human coronary atherosclerosis progressed?Myeloid CD34+CD13+ precursor cells transdifferentiate into chondrocyte-like cells in atherosclerotic intimal calcification Runx2/Cbfa1, but not loss of myocardin, is required for smooth muscle cell lineage reprogramming toward osteochondrogenesis.Calcification after myocardial infarction is independent of amniotic fluid stem cell injection.Vitamin K-antagonists accelerate atherosclerotic calcification and induce a vulnerable plaque phenotype.PKA-induced receptor activator of NF-kappaB ligand (RANKL) expression in vascular cells mediates osteoclastogenesis but not matrix calcification Vascular smooth muscle cells ablation with endovascular nonthermal irreversible electroporation Arterial calcification: Finger-pointing at resident and circulating stem cells.Age Is the Strongest Effector for the Relationship between Estimated Glomerular Filtration Rate and Coronary Artery Calcification in Apparently Healthy Korean Adults.Inhibition of bone morphogenetic proteins protects against atherosclerosis and vascular calcification.Reducing Jagged 1 and 2 levels prevents cerebral arteriovenous malformations in matrix Gla protein deficiency.Physiopathology of intratendinous calcific deposition Overnutrition, mTOR signaling, and cardiovascular diseases.Targeted reduction of vascular Msx1 and Msx2 mitigates arteriosclerotic calcification and aortic stiffness in LDLR-deficient mice fed diabetogenic diets.Well-organized neointima of large-pore poly(L-lactic acid) vascular graft coated with poly(L-lactic-co-ε-caprolactone) prevents calcific deposition compared to small-pore electrospun poly(L-lactic acid) graft in a mouse aortic implantation model Arterial injury promotes medial chondrogenesis in Sm22 knockout mice.Decreased microRNA is involved in the vascular remodeling abnormalities in chronic kidney disease (CKD).Elastin degradation and vascular smooth muscle cell phenotype change precede cell loss and arterial medial calcification in a uremic mouse model of chronic kidney disease.

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Smooth muscle cells give rise to osteochondrogenic precursors and chondrocytes in calcifying arteries.

http://www.wikidata.org/entity/Q37276028

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 05 February 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Smooth muscle cells give rise ...... ocytes in calcifying arteries.

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Smooth muscle cells give rise ...... ocytes in calcifying arteries.

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type

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Smooth muscle cells give rise ...... ocytes in calcifying arteries.

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Smooth muscle cells give rise ...... ocytes in calcifying arteries.

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prefLabel

Smooth muscle cells give rise ...... ocytes in calcifying arteries.

@en

Smooth muscle cells give rise ...... ocytes in calcifying arteries.

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CIRCRESAHA.108.183053

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Circulation Research

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Smooth muscle cells give rise ...... ocytes in calcifying arteries.

@en

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Amy Look

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Andrew Frutkin

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Cecilia M Giachelli

http://www.w3.org/2001/XMLSchema#string

David Dichek

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Elizabeth Leaf

http://www.w3.org/2001/XMLSchema#string

Hsueh-Ying Yang

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Mei Y Speer

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Thea Brabb

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Wei-Ling Lin

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P2860

Mutations in the gene encoding the human matrix Gla protein cause Keutel syndrome

Atherosclerosis — An Inflammatory Disease

Generalized lacZ expression with the ROSA26 Cre reporter strain

Spontaneous calcification of arteries and cartilage in mice lacking matrix GLA protein

Canonical Wnt/beta-catenin signaling prevents osteoblasts from differentiating into chondrocytes

Matrix Gla protein accumulates at the border of regions of calcification and normal tissue in the media of the arterial vessel wall

LRP: role in vascular wall integrity and protection from atherosclerosis.

Mutations in ENPP1 are associated with 'idiopathic' infantile arterial calcification.

Myocardin and ternary complex factors compete for SRF to control smooth muscle gene expression.

Control of serum phosphorus: implications for coronary artery calcification and calcific uremic arteriolopathy (calciphylaxis).

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10.1161/CIRCRESAHA.108.183053

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6

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104

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2009-02-05T00:00:00Z

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19197075

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2716055

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