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Bile acids: regulation of apoptosis by ursodeoxycholic acidBile acids: regulation of synthesisBile acids: emerging role in management of liver diseasesImproved glucose metabolism following bariatric surgery is associated with increased circulating bile acid concentrations and remodeling of the gut microbiomeThe role of bile salts in liver regenerationPlasma metabolomic profile in nonalcoholic fatty liver disease.Waking action of ursodeoxycholic acid (UDCA) involves histamine and GABAA receptor block.Bile acids induce hepatic differentiation of mesenchymal stem cellsTGR5 potentiates GLP-1 secretion in response to anionic exchange resins.Role of nuclear receptors in lipid dysfunction and obesity-related diseases.Comparison of Cytotoxic Activity of Bile on HepG2 and CCRF-CEM Cell Lines: An in Vitro Study.Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME.Excretion of biliary compounds during intrauterine life.Biosynthesis and trafficking of the bile salt export pump, BSEP: therapeutic implications of BSEP mutations.Targeting nuclear bile acid receptors for liver disease.The therapeutic potential of manipulating gut microbiota in obesity and type 2 diabetes mellitus.Anticancer steroids: linking natural and semi-synthetic compounds.The bile acid membrane receptor TGR5: a novel pharmacological target in metabolic, inflammatory and neoplastic disorders.The bile acid TGR5 membrane receptor: from basic research to clinical application.Cholestatic liver (dys)function during sepsis and other critical illnesses.A shift in paradigm towards human biology-based systems for cholestatic-liver diseases.Bile acids and bile acid derivatives: use in drug delivery systems and as therapeutic agents.TGR5 is essential for bile acid-dependent cholangiocyte proliferation in vivo and in vitro.Expression and function of the bile acid receptor GpBAR1 (TGR5) in the murine enteric nervous system.Bile Acids Act as Soluble Host Restriction Factors Limiting Cytomegalovirus Replication in Hepatocytes.Safety, Pharmacokinetics, and Pharmacodynamic Effects of a Selective TGR5 Agonist, SB-756050, in Type 2 Diabetes.Allelic variation of bile salt hydrolase genes in Lactobacillus salivarius does not determine bile resistance levels.Role of the bile acid receptor TGR5 (Gpbar-1) in liver damage and regeneration.Changes of the plasma metabolome during an oral glucose tolerance test: is there more than glucose to look at?Reprogramming of pro-inflammatory human macrophages to an anti-inflammatory phenotype by bile acids.On the Role of Illness Duration and Nutrient Restriction in Cholestatic Alterations that Occur During Critical Illness.High prevalence of cholestasis, with increased conjugated bile acids in inflammatory bowel diseases patients.Anti-inflammatory consequences of bile acid accumulation in virus-infected bile duct ligated mice.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Endocrine and paracrine role of bile acids
@en
Endocrine and paracrine role of bile acids.
@nl
type
label
Endocrine and paracrine role of bile acids
@en
Endocrine and paracrine role of bile acids.
@nl
prefLabel
Endocrine and paracrine role of bile acids
@en
Endocrine and paracrine role of bile acids.
@nl
P2860
P356
P1476
Endocrine and paracrine role of bile acids
@en
P2093
Ralf Kubitz
Verena Keitel
P2860
P304
P356
10.3748/WJG.14.5620
P407
P577
2008-10-01T00:00:00Z