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Macrophage and T cell produced IL-10 promotes viral chronicity

Macrophage and T cell produced IL-10 promotes viral chronicity

http://www.wikidata.org/entity/Q37288927

about

Molecular and cellular insights into T cell exhaustion The role of myeloid cell activation and arginine metabolism in the pathogenesis of virus-induced diseases Viral Infection of the Central Nervous System Exacerbates Interleukin-10 Receptor Deficiency-Mediated Colitis in SJL Mice Chronic viral infection promotes sustained Th1-derived immunoregulatory IL-10 via BLIMP-1 Early virus-host interactions dictate the course of a persistent infection Direct infection of dendritic cells during chronic viral infection suppresses antiviral T cell proliferation and induces IL-10 expression in CD4 T cells.T cell exhaustion during persistent viral infections.Macrophage Polarization in Virus-Host Interactions Type I and Type II Interferon Coordinately Regulate Suppressive Dendritic Cell Fate and Function during Viral Persistence The role of type I interferons in intestinal infection, homeostasis, and inflammation.Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.HIV Progression Perturbs the Balance of the Cell-Mediated and Anti-Inflammatory Adaptive and Innate Mycobacterial Immune Response.Exploitation of Interleukin-10 (IL-10) Signaling Pathways: Alternate Roles of Viral and Cellular IL-10 in Rhesus Cytomegalovirus Infection.Regulator Versus Effector Paradigm: Interleukin-10 as Indicator of the Switching Response.IL-10: A Multifunctional Cytokine in Viral Infections.HLA-G+ HIV-1-specific CD8+ T cells are associated with HIV-1 immune control.IL-10 Regulates Memory T Cell Development and the Balance between Th1 and Follicular Th Cell Responses during an Acute Viral Infection.Schistosome Soluble Egg Antigen Decreases Mycobacterium tuberculosis-Specific CD4+ T-Cell Effector Function With Concomitant Arrest of Macrophage Phago-Lysosome Maturation.Suppressive effect of interleukin 10 on priming of naive hepatitis C virus-specific CD8+ T cells.T cell derived IL-10 is dispensable for tolerance induction in a murine model of allergic airway inflammation.Intact interleukin-10 receptor signaling protects from hippocampal damage elicited by experimental neurotropic virus infection of SJL mice.

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Macrophage and T cell produced IL-10 promotes viral chronicity

http://www.wikidata.org/entity/Q37288927

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

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2013年學術文章

@zh-hant

name

Macrophage and T cell produced IL-10 promotes viral chronicity

@en

Macrophage and T cell produced IL-10 promotes viral chronicity.

@nl

type

label

Macrophage and T cell produced IL-10 promotes viral chronicity

@en

Macrophage and T cell produced IL-10 promotes viral chronicity.

@nl

prefLabel

Macrophage and T cell produced IL-10 promotes viral chronicity

@en

Macrophage and T cell produced IL-10 promotes viral chronicity.

@nl

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JOURNAL.PPAT.1003735

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Macrophage and T cell produced IL-10 promotes viral chronicity

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Axel Roers

http://www.w3.org/2001/XMLSchema#string

Guillaume Perriard

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Kirsten Richter

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Masahito Kamanaka

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Reto A Schwendener

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Richard A Flavell

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Robert Dunn

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Veronika Sexl

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P2860

Programmed death-1-induced interleukin-10 production by monocytes impairs CD4+ T cell activation during HIV infection

Interleukin-10 determines viral clearance or persistence in vivo

Expression of interleukin-10 in intestinal lymphocytes detected by an interleukin-10 reporter knockin tiger mouse

Interleukin-10-deficient mice develop chronic enterocolitis

In vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigens

The regulation of IL-10 production by immune cells

Two types of mouse T helper cell. IV. Th2 clones secrete a factor that inhibits cytokine production by Th1 clones

Virus-encoded homologs of cellular interleukin-10 and their control of host immune function.

IL-10 inhibits macrophage activation and proliferation by distinct signaling mechanisms: evidence for Stat3-dependent and -independent pathways

Posttranscriptional regulation of IL-10 gene expression through sequences in the 3'-untranslated region.

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e1003735

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scholarly article

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10.1371/JOURNAL.PPAT.1003735

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11

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9

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Annette Oxenius

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2013-11-07T00:00:00Z

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258639197

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24244162

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3820745

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