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Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

http://www.wikidata.org/entity/Q37303806

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Klf15 deficiency is a molecular link between heart failure and aortic aneurysm formation Metabogenic and Nutriceutical Approaches to Address Energy Dysregulation and Skeletal Muscle Wasting in Duchenne Muscular Dystrophy Structure of the MADS-box/MEF2 Domain of MEF2A Bound to DNA and Its Implication for Myocardin Recruitment Structure of p300 bound to MEF2 on DNA reveals a mechanism of enhanceosome assembly Epigenetic regulation of endothelial-cell-mediated vascular repair Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophy Auto-acetylation stabilizes p300 in cardiac myocytes during acute oxidative stress, promoting STAT3 accumulation and cell survival Transcriptional/epigenetic regulator CBP/p300 in tumorigenesis: structural and functional versatility in target recognition.Epigenetic mechanisms in heart failure pathogenesis BET bromodomains mediate transcriptional pause release in heart failure Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling.Calcium-mediated histone modifications regulate alternative splicing in cardiomyocytes.Role of apoptosis signal-regulating kinase 1 (ASK1) as an activator of the GAPDH-Siah1 stress-signaling cascade.Cooperative activation of Npr1 gene transcription and expression by interaction of Ets-1 and p300.MicroRNA-20a constrains p300-driven myocardial angiogenic transcription by direct targeting of p300.KLF15 and PPARα Cooperate to Regulate Cardiomyocyte Lipid Gene Expression and Oxidation.Gene expression network analysis reveals new transcriptional regulators as novel factors in human ischemic cardiomyopathy Roles and post-translational regulation of cardiac class IIa histone deacetylase isoforms.Bnip3 Binds and Activates p300: Possible Role in Cardiac Transcription and Myocyte Morphology.The histone 3 lysine 9 methyltransferase inhibitor chaetocin improves prognosis in a rat model of high salt diet-induced heart failure.The virtue of just enough stress: a molecular model.It's Time for An Epigenomics Roadmap of Heart Failure Resveratrol improves cardiomyopathy in dystrophin-deficient mice through SIRT1 protein-mediated modulation of p300 protein.Genetics and epigenetics of arrhythmia and heart failure.SIRT1 deacetylates the cardiac transcription factor Nkx2.5 and inhibits its transcriptional activity.Kruppel-like factor 15 is a critical regulator of cardiac lipid metabolism Mapping cell type-specific transcriptional enhancers using high affinity, lineage-specific Ep300 bioChIP-seq.Molecular mechanisms of cardiomyocyte aging.Understanding cardiovascular disease: a journey through the genome (and what we found there).Glucose-induced cell signaling in the pathogenesis of diabetic cardiomyopathy.Epigenetic regulation and heart failure.Epigenetics of the failing heart.Epigenetic modifications and noncoding RNAs in cardiac hypertrophy and failure.The effects of resveratrol and SIRT1 activation on dystrophic cardiomyopathy.Elucidating the mechanisms of transcription regulation during heart development by next-generation sequencing.Epigenetic regulation of cardiac fibrosis.MicroRNA-142 is a multifaceted regulator in organogenesis, homeostasis, and disease.Phenylephrine-induced cardiac hypertrophy is attenuated by a histone acetylase inhibitor anacardic acid in mice.Metabolic Reprogramming via PPARα Signaling in Cardiac Hypertrophy and Failure From Metabolomics to Epigenetics.Molecular pathways underlying cardiac remodeling during pathophysiological stimulation.

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Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

http://www.wikidata.org/entity/Q37303806

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 12 August 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@en

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@nl

type

label

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@en

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@nl

prefLabel

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@en

Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

@nl

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CIRCULATIONAHA.107.760488

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Quantitative control of adaptive cardiac hypertrophy by acetyltransferase p300.

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James M Mitrani

http://www.w3.org/2001/XMLSchema#string

Jian Qin Wei

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Keith A Webster

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Lina A Shehadeh

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Monica Pessanha

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Nanette H Bishopric

http://www.w3.org/2001/XMLSchema#string

Tatiana I Slepak

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P2860

Nuclear Rho kinase, ROCK2, targets p300 acetyltransferase

Conditional knockout mice reveal distinct functions for the global transcriptional coactivators CBP and p300 in T-cell development

Molecular mechanisms of myogenic coactivation by p300: direct interaction with the activation domain of MyoD and with the MADS box of MEF2C

Histone deacetylase 3 interacts with and deacetylates myocyte enhancer factor 2

Control of cardiac-specific transcription by p300 through myocyte enhancer factor-2D

CREB-binding protein and p300 in transcriptional regulation

Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy

Gene dosage-dependent embryonic development and proliferation defects in mice lacking the transcriptional integrator p300

Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload

Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activity

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934-946

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scholarly article

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10.1161/CIRCULATIONAHA.107.760488

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2008-08-12T00:00:00Z

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18697823

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