Involvement of tumor necrosis factor-alpha in angiotensin II-mediated effects on salt appetite, hypertension, and cardiac hypertrophy.
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Cytomegalovirus infection causes an increase of arterial blood pressureInflammation, immunity, and hypertensive end-organ damageSalt-Sensitive Hypertension: Perspectives on Intrarenal MechanismsThe inextricable role of the kidney in hypertensionInhibition of inflammation and oxidative stress by an imidazopyridine derivative X22 prevents heart injury from obesityIncreased circulating ANG II and TNF-α represents important risk factors in obese saudi adults with hypertension irrespective of diabetic status and BMITumor necrosis factor α decreases nitric oxide synthase type 3 expression primarily via Rho/Rho kinase in the thick ascending limbConstitutive smooth muscle tumour necrosis factor regulates microvascular myogenic responsiveness and systemic blood pressureTumor necrosis factor-α produced in the kidney contributes to angiotensin II-dependent hypertension.Caveolin 1 is critical for abdominal aortic aneurysm formation induced by angiotensin II and inhibition of lysyl oxidase.Interleukin 17 promotes angiotensin II-induced hypertension and vascular dysfunction.Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets?NF-kappaB-induced oxidative stress contributes to mitochondrial and cardiac dysfunction in type II diabetes.Roles of inflammation, oxidative stress, and vascular dysfunction in hypertension.Brain microglial cytokines in neurogenic hypertensionHDAC inhibition attenuates inflammatory, hypertrophic, and hypertensive responses in spontaneously hypertensive rats.Angiotensin II-induced upregulation of AT(1) receptor expression: sequential activation of NF-kappaB and Elk-1 in neurons.TNFR1-deficient mice display altered blood pressure and renal responses to ANG II infusion.Interaction of TNF with angiotensin II contributes to mitochondrial oxidative stress and cardiac damage in ratsInhibition of TNF in the brain reverses alterations in RAS components and attenuates angiotensin II-induced hypertensionNatural regulatory T cells control coronary arteriolar endothelial dysfunction in hypertensive miceParaventricular nucleus corticotrophin releasing hormone contributes to sympathoexcitation via interaction with neurotransmitters in heart failure.Tumor necrosis factor-alpha is an endogenous inhibitor of Na+-K+-2Cl- cotransporter (NKCC2) isoform A in the thick ascending limb.Toll-like receptor 4 inhibition within the paraventricular nucleus attenuates blood pressure and inflammatory response in a genetic model of hypertensionTumor necrosis factor: a mechanistic link between angiotensin-II-induced cardiac inflammation and fibrosis.Role of T lymphocytes in hypertension.Lymphocyte responses exacerbate angiotensin II-dependent hypertension.Chronic exercise modulates RAS components and improves balance between pro- and anti-inflammatory cytokines in the brain of SHRIncreased angiotensin II-induced hypertension and inflammatory cytokines in mice lacking angiotensin-converting enzyme N domain activity.Angiotensin II-induced hypertension is modulated by nuclear factor-κBin the paraventricular nucleus.ACE2 overexpression in the paraventricular nucleus attenuates angiotensin II-induced hypertensionTumor Necrosis Factor - Alpha Is Essential for Angiotensin II-Induced Ventricular Remodeling: Role for Oxidative Stress.Adiponectin Upregulates MiR-133a in Cardiac Hypertrophy through AMPK Activation and Reduced ERK1/2 PhosphorylationDual Activation of TRIF and MyD88 Adaptor Proteins by Angiotensin II Evokes Opposing Effects on Pressure, Cardiac Hypertrophy, and Inflammatory Gene Expression.Tumor necrosis factor-α receptor type 1, not type 2, mediates its acute responses in the kidneyA novel role for type 1 angiotensin receptors on T lymphocytes to limit target organ damage in hypertension.Effect of psoriasis severity on hypertension control: a population-based study in the United KingdomThe role of type 1 angiotensin receptors on T lymphocytes in cardiovascular and renal diseases.TNF-α type 2 receptor mediates renal inflammatory response to chronic angiotensin II administration with high salt intake in mice.Salt Sensitivity in Response to Renal Injury Requires Renal Angiotensin-Converting Enzyme
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P2860
Involvement of tumor necrosis factor-alpha in angiotensin II-mediated effects on salt appetite, hypertension, and cardiac hypertrophy.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 07 April 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@en
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@nl
type
label
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@en
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@nl
prefLabel
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@en
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@nl
P2093
P2860
P921
P1433
P1476
Involvement of tumor necrosis ...... sion, and cardiac hypertrophy.
@en
P2093
Dewan S A Majid
Joseph Francis
Masudul Haque
P2860
P304
P356
10.1161/HYPERTENSIONAHA.107.102152
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P577
2008-04-07T00:00:00Z