about
NF-kappaB-induced oxidative stress contributes to mitochondrial and cardiac dysfunction in type II diabetes.Brain microglial cytokines in neurogenic hypertensionAngiotensin II mediates angiotensin converting enzyme type 2 internalization and degradation through an angiotensin II type I receptor-dependent mechanism.Inhibition of TNF in the brain reverses alterations in RAS components and attenuates angiotensin II-induced hypertensionACE2/ANG-(1-7)/Mas pathway in the brain: the axis of good.Brain-targeted angiotensin-converting enzyme 2 overexpression attenuates neurogenic hypertension by inhibiting cyclooxygenase-mediated inflammation.Brain ACE2 overexpression reduces DOCA-salt hypertension independently of endoplasmic reticulum stress.Species-specific inhibitor sensitivity of angiotensin-converting enzyme 2 (ACE2) and its implication for ACE2 activity assaysCentral TNF inhibition results in attenuated neurohumoral excitation in heart failure: a role for superoxide and nitric oxide.Angiotensin II-induced hypertension is modulated by nuclear factor-κBin the paraventricular nucleus.ACE2 overexpression in the paraventricular nucleus attenuates angiotensin II-induced hypertensionBrain angiotensin-converting enzyme type 2 shedding contributes to the development of neurogenic hypertension.Tumor Necrosis Factor - Alpha Is Essential for Angiotensin II-Induced Ventricular Remodeling: Role for Oxidative Stress.Evidence for angiotensin-converting enzyme 2 as a therapeutic target for the prevention of pulmonary hypertension.Prevention of pulmonary hypertension by Angiotensin-converting enzyme 2 gene transfer.Involvement of tumor necrosis factor-alpha in angiotensin II-mediated effects on salt appetite, hypertension, and cardiac hypertrophy.Clinical Relevance and Role of Neuronal AT1 Receptors in ADAM17-Mediated ACE2 Shedding in Neurogenic Hypertension.Kinin B1 Receptor Promotes Neurogenic Hypertension Through Activation of Centrally Mediated Mechanisms.Determining the Enzymatic Activity of Angiotensin-Converting Enzyme 2 (ACE2) in Brain Tissue and Cerebrospinal Fluid Using a Quenched Fluorescent Substrate.Excessive Glutamate Stimulation Impairs ACE2 Activity Through ADAM17-Mediated Shedding in Cultured Cortical Neurons.Glutamatergic neurons of the paraventricular nucleus are critical contributors to the development of neurogenic hypertensionTNF-alpha-induced mitochondrial oxidative stress and cardiac dysfunction: restoration by superoxide dismutase mimetic TempolAging influences cerebrovascular myogenic reactivity and BK channel function in a sex-specific mannerKinin B1 Receptor Blockade Prevents Angiotensin II-induced Neuroinflammation and Oxidative Stress in Primary Hypothalamic Neurons
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P50
description
Forscher
@de
chercheur
@fr
investigador
@es
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
研究者
@zh
name
Srinivas Sriramula
@ast
Srinivas Sriramula
@en
Srinivas Sriramula
@es
Srinivas Sriramula
@nl
type
label
Srinivas Sriramula
@ast
Srinivas Sriramula
@en
Srinivas Sriramula
@es
Srinivas Sriramula
@nl
prefLabel
Srinivas Sriramula
@ast
Srinivas Sriramula
@en
Srinivas Sriramula
@es
Srinivas Sriramula
@nl
P1153
22987066900
P31
P496
0000-0001-9937-1589