Inactivation of uPA and its receptor uPAR by 3,3'-diindolylmethane (DIM) leads to the inhibition of prostate cancer cell growth and migration.
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Cellular and Molecular Mechanisms of 3,3'-Diindolylmethane in Gastrointestinal CancerRoles of Dietary Phytoestrogens on the Regulation of Epithelial-Mesenchymal Transition in Diverse Cancer MetastasisNatural compounds as anticancer agents: Experimental evidenceRecent progress on nutraceutical research in prostate cancer.Attenuation of multi-targeted proliferation-linked signaling by 3,3'-diindolylmethane (DIM): from bench to clinic.Novel targets for prostate cancer chemoprevention.Multiple therapeutic and preventive effects of 3,3'-diindolylmethane on cancers including prostate cancer and high grade prostatic intraepithelial neoplasia.Elongation factor-2 kinase regulates TG2/β1 integrin/Src/uPAR pathway and epithelial-mesenchymal transition mediating pancreatic cancer cells invasionMechanisms and functional consequences of PDEF protein expression loss during prostate cancer progression.3'3-Diindolylmethane inhibits migration, invasion and metastasis of hepatocellular carcinoma by suppressing FAK signaling.Down-regulation of uPA and uPAR by 3,3'-diindolylmethane contributes to the inhibition of cell growth and migration of breast cancer cellsMechanisms and therapeutic implications of cell death induction by indole compounds.Advances in androgen receptor targeted therapy for prostate cancerSystems and Network Pharmacology Approaches to Cancer Stem Cells Research and Therapy.PAR-4 as a possible new target for pancreatic cancer therapy.A novel peptide blocking cancer cell invasion by structure-based drug design.Effect of diindolylmethane on Ca2+ homeostasis and viability in PC3 human prostate cancer cells.ProstaCaid induces G2/M cell cycle arrest and apoptosis in human and mouse androgen-dependent and-independent prostate cancer cells.FoxM1 down-regulation leads to inhibition of proliferation, migration and invasion of breast cancer cells through the modulation of extra-cellular matrix degrading factors.Spontaneous regression of septum pellucidum/forniceal pilocytic astrocytomas--possible role of Cannabis inhalation.
P2860
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P2860
Inactivation of uPA and its receptor uPAR by 3,3'-diindolylmethane (DIM) leads to the inhibition of prostate cancer cell growth and migration.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on June 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Inactivation of uPA and its re ...... cer cell growth and migration.
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Inactivation of uPA and its re ...... cer cell growth and migration.
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type
label
Inactivation of uPA and its re ...... cer cell growth and migration.
@en
Inactivation of uPA and its re ...... cer cell growth and migration.
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prefLabel
Inactivation of uPA and its re ...... cer cell growth and migration.
@en
Inactivation of uPA and its re ...... cer cell growth and migration.
@nl
P2093
P2860
P356
P1476
Inactivation of uPA and its re ...... cer cell growth and migration.
@en
P2093
Aamir Ahmad
Dejuan Kong
Fazlul H Sarkar
Sanila H Sarkar
Sanjeev Banerjee
Zhiwei Wang
P2860
P304
P356
10.1002/JCB.22152
P577
2009-06-01T00:00:00Z