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Genetic background affects susceptibility to tumoral stem cell reprogramming.

Genetic background affects susceptibility to tumoral stem cell reprogramming.

http://www.wikidata.org/entity/Q37344811

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Genomic quantitative real-time PCR proves residual disease positivity in more than 30% samples with negative mRNA-based qRT-PCR in Chronic Myeloid Leukemia Lineage-specific function of Engrailed-2 in the progression of chronic myelogenous leukemia to T-cell blast crisis.

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Genetic background affects susceptibility to tumoral stem cell reprogramming.

http://www.wikidata.org/entity/Q37344811

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 July 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Genetic background affects susceptibility to tumoral stem cell reprogramming.

@en

Genetic background affects susceptibility to tumoral stem cell reprogramming.

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type

label

Genetic background affects susceptibility to tumoral stem cell reprogramming.

@en

Genetic background affects susceptibility to tumoral stem cell reprogramming.

@nl

prefLabel

Genetic background affects susceptibility to tumoral stem cell reprogramming.

@en

Genetic background affects susceptibility to tumoral stem cell reprogramming.

@nl

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Genetic background affects susceptibility to tumoral stem cell reprogramming

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Francisco Javier García-Criado

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Idoia García-Ramírez

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Lucía Ruiz-Roca

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Germinal centre protein HGAL promotes lymphoid hyperplasia and amyloidosis via BCR-mediated Syk activation

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Bcl2 is not required for the development and maintenance of leukemia stem cells in mice.

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Persistence of leukemia stem cells in chronic myelogenous leukemia patients in prolonged remission with imatinib treatment

Chronic myeloid leukemia stem cells are not dependent on Bcr-Abl kinase activity for their survival

Essential role for telomerase in chronic myeloid leukemia induced by BCR-ABL in mice.

Expression of MALT1 oncogene in hematopoietic stem/progenitor cells recapitulates the pathogenesis of human lymphoma in mice.

Loss of p53 exacerbates multiple myeloma phenotype by facilitating the reprogramming of hematopoietic stem/progenitor cells to malignant plasma cells by MafB.

Cancer induction by restriction of oncogene expression to the stem cell compartment

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2505-2509

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scholarly article

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10.4161/CC.25544

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15

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12

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Carolina Vicente-Dueñas

Isidro Sanchez-garcia

Alberto Martín-Lorenzo

María Begoña García Cenador

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2013-07-08T00:00:00Z

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248384419

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23839033

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3841328

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