Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
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How Useful Are Monogenic Rodent Models for the Study of Human Non-Alcoholic Fatty Liver Disease?Constitutive Activation of AKT2 in Humans Leads to Hypoglycemia Without Fatty Liver or Metabolic Dyslipidemia.Domain analysis reveals striking functional differences between the regulatory subunits of phosphatidylinositol 3-kinase (PI3K), p85α and p85β.Treatment of a case of severe insulin resistance as a result of a PIK3R1 mutation with a sodium-glucose cotransporter 2 inhibitor.Molecular Mechanisms of Human Disease Mediated by Oncogenic and Primary Immunodeficiency Mutations in Class IA Phosphoinositide 3-Kinases.
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Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 20 October 2016
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vedecký článok
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vetenskaplig artikel
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Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@en
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@nl
type
label
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@en
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@nl
prefLabel
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@en
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@nl
P2093
P2860
P50
P1433
P1476
Insulin resistance uncoupled from dyslipidemia due to C-terminal PIK3R1 mutations.
@en
P2093
Albert Kwok
Alexandra Gast
Allan Daly
Barbara Müksch
Emma McCann
Jonathan Clark
Julie Harris
Lazaros C Foukas
Mehul Dattani
Nuno Rocha
P2860
P304
P356
10.1172/JCI.INSIGHT.88766
P577
2016-10-20T00:00:00Z