Bioavailable copper modulates oxidative phosphorylation and growth of tumors. - Wikidata - wikimedia - TriplyDB

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

http://www.wikidata.org/entity/Q37353035

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Molecular imaging and therapy targeting copper metabolism in hepatocellular carcinoma Synthetic fluorescent probes for studying copper in biological systems Tetracycline antibiotics impair mitochondrial function and its experimental use confounds research Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge ahead Copper activates HIF-1α/GPER/VEGF signalling in cancer cells Roles of Copper-Binding Proteins in Breast Cancer.Natural variations of copper and sulfur stable isotopes in blood of hepatocellular carcinoma patients.Copper depletion inhibits CoCl2-induced aggressive phenotype of MCF-7 cells via downregulation of HIF-1 and inhibition of Snail/Twist-mediated epithelial-mesenchymal transition.Chemotherapy triggers HIF-1-dependent glutathione synthesis and copper chelation that induces the breast cancer stem cell phenotype.Tetrathiomolybdate inhibits mitochondrial complex IV and mediates degradation of hypoxia-inducible factor-1α in cancer cells.Copper-64 Dichloride as Theranostic Agent for Glioblastoma Multiforme: A Preclinical Study.Inhibition of human copper trafficking by a small molecule significantly attenuates cancer cell proliferation Disulfiram anti-cancer efficacy without copper overload is enhanced by extracellular H2O2 generation: antagonism by tetrathiomolybdate.Targeting drug transport mechanisms for improving platinum-based cancer chemotherapy.Transcriptome analysis of copper homeostasis genes reveals coordinated upregulation of SLC31A1,SCO1, and COX11 in colorectal cancer.In vivo bioluminescence imaging reveals copper deficiency in a murine model of nonalcoholic fatty liver disease.Characterization and use of HapT1-derived homologous tumors as a preclinical model to evaluate therapeutic efficacy of drugs against pancreatic tumor desmoplasia.Mechanisms of base substitution mutagenesis in cancer genomes.Oxidation-Induced Degradable Nanogels for Iron Chelation Targeting copper in cancer therapy: 'Copper That Cancer'.Copper and conquer: copper complexes of di-2-pyridylketone thiosemicarbazones as novel anti-cancer therapeutics.Medical applications of Cu, Zn, and S isotope effects.Lysyl oxidase isoforms in gastric cancer.Time-resolved ICP-MS analysis of mineral element contents and distribution patterns in single cells.Effects of copper on CHO cells: cellular requirements and product quality considerations.Analytical Methods for Imaging Metals in Biology: From Transition Metal Metabolism to Transition Metal Signaling.Copper Ion from Cu2O Crystal Induces AMPK-Mediated Autophagy via Superoxide in Endothelial Cells.Transcriptional Responses and Mechanisms of Copper-Induced Dysfunctional Locomotor Behavior in Zebrafish Embryos.A Role for The ATP7A Copper Transporter in Tumorigenesis and Cisplatin Resistance Effect of Copper Sulfate on Expression of Endogenous L1 Retrotransposons in HepG2 Cells (Hepatocellular Carcinoma).Selective elimination of neuroblastoma cells by synergistic effect of Akt kinase inhibitor and tetrathiomolybdate.CD147 functions as the signaling receptor for extracellular divalent copper in hepatocellular carcinoma cells.Copper isotope effect in serum of cancer patients. A pilot study.Peptides derived from histidine and methionine-rich regions of copper transporter 1 exhibit anti-angiogenic property by chelating extracellular Cu.Improving lactate metabolism in an intensified CHO culture process: productivity and product quality considerations.Copper(II), cobalt(II) and nickel(II) complexes of juglone: synthesis, structure, DNA interaction and enhanced cytotoxicity.Effects of Terminal Substitution and Iron Coordination on Antiproliferative Activity of l -Proline-salicylaldehyde-Thiosemicarbazone Hybrids Cu selective chelators relieve copper-induced oxidative stress

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P2860

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

http://www.wikidata.org/entity/Q37353035

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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2013年學術文章

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name

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

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Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

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type

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Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

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Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

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prefLabel

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

@en

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

@nl

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PNAS.1318431110

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Bioavailable copper modulates oxidative phosphorylation and growth of tumors.

@en

P2093

Carole Poitry-Yamate

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Douglas Hanahan

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Seiko Ishida

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P2860

Understanding the Warburg effect: the metabolic requirements of cell proliferation

Energy metabolism in tumor cells

Regulation of cancer cell metabolism

Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

Evidence that metformin exerts its anti-diabetic effects through inhibition of complex 1 of the mitochondrial respiratory chain

AMPK: a nutrient and energy sensor that maintains energy homeostasis

Cohesin: its roles and mechanisms

Metformin and reduced risk of cancer in diabetic patients

On respiratory impairment in cancer cells

Beta-cell lines derived from transgenic mice expressing a hybrid insulin gene-oncogene.

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19507-19512

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10.1073/PNAS.1318431110

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48

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110

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Pénélope Andreux

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2013-11-11T00:00:00Z

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258446198

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phosphorylation

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