Direct chronic effect of steroid hormones in attenuating uterine arterial myogenic tone: role of protein kinase c/extracellular signal-regulated kinase 1/2.

Direct chronic effect of steroid hormones in attenuating uterine arterial myogenic tone: role of protein kinase c/extracellular signal-regulated kinase 1/2.

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http://www.wikidata.org/entity/Q37355263
Gestational hypoxia up-regulates protein kinase C and inhibits calcium-activated potassium channels in ovine uterine arteries.Higher estrogen levels during pregnancy in Andean than European residents of high altitude suggest differences in aromatase activityChronic hypoxia inhibits sex steroid hormone-mediated attenuation of ovine uterine arterial myogenic tone in pregnancy.Pregnancy downregulates actin polymerization and pressure-dependent myogenic tone in ovine uterine arteries.Chronic hypoxia during gestation enhances uterine arterial myogenic tone via heightened oxidative stress.PKC regulates alpha(1)-adrenoceptor-mediated contractions and baseline Ca(2+) sensitivity in the uterine arteries of nonpregnant and pregnant sheep acclimatized to high altitude hypoxia.Gestational hypoxia increases reactive oxygen species and inhibits steroid hormone-mediated upregulation of Ca(2+)-activated K(+) channel function in uterine arteries.Progesterone interactions with the cervix: translational implications for term and preterm birth.Pregnancy upregulates large-conductance Ca(2+)-activated K(+) channel activity and attenuates myogenic tone in uterine arteries.Estrogen Replacement Therapy in Ovariectomized Nonpregnant Ewes Stimulates Uterine Artery Hydrogen Sulfide Biosynthesis by Selectively Up-Regulating Cystathionine β-Synthase Expression.Chronic hypoxia differentially up-regulates protein kinase C-mediated ovine uterine arterial contraction via actin polymerization signaling in pregnancy.Hypoxia Represses ER-α Expression and Inhibits Estrogen-Induced Regulation of Ca2+-Activated K+ Channel Activity and Myogenic Tone in Ovine Uterine Arteries: Causal Role of DNA MethylationChronic hypoxia during gestation causes epigenetic repression of the estrogen receptor-α gene in ovine uterine arteries via heightened promoter methylation.Chronic hypoxia suppresses pregnancy-induced upregulation of large-conductance Ca2+-activated K+ channel activity in uterine arteriesChronic hypoxia inhibits pregnancy-induced upregulation of SKCa channel expression and function in uterine arteries.Prenatal cocaine exposure causes sex-dependent impairment in the myogenic reactivity of coronary arteries in adult offspring.Direct effect of chronic hypoxia in suppressing large conductance Ca(2+)-activated K(+) channel activity in ovine uterine arteries via increasing oxidative stress.
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Direct chronic effect of steroid hormones in attenuating uterine arterial myogenic tone: role of protein kinase c/extracellular signal-regulated kinase 1/2.

Item
http://www.wikidata.org/entity/Q37355263
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 15 June 2009
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
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Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
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type
label
Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
@en
Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
@nl
prefLabel
Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
@en
Direct chronic effect of stero ...... r signal-regulated kinase 1/2.
@nl
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Direct chronic effect of stero ...... ar signal-regulated kinase 1/2
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10.1161/HYPERTENSIONAHA.109.130781
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P433
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