Transforming growth factor-beta1 (TGF-beta1) induces human osteoclast apoptosis by up-regulating Bim. - Wikidata - wikimedia - TriplyDB

Transforming growth factor-beta1 (TGF-beta1) induces human osteoclast apoptosis by up-regulating Bim.

Transforming growth factor-beta1 (TGF-beta1) induces human osteoclast apoptosis by up-regulating Bim.

http://www.wikidata.org/entity/Q37358112

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Regulation of Bim in Health and Disease Activin A inhibits RANKL-mediated osteoclast formation, movement and function in murine bone marrow macrophage cultures.Loss of the Birt-Hogg-Dubé tumor suppressor results in apoptotic resistance due to aberrant TGFβ-mediated transcription Role of TGF-β in a mouse model of high turnover renal osteodystrophy.Effects of LLLT in combination with bisphosphonate on bone healing in critical size defects: a histological and histometric study in rat calvaria.Characterization and identification of subpopulations of mononuclear preosteoclasts induced by TNF-α in combination with TGF-β in rats.The molecular mechanisms of regulatory T cell immunosuppression Targeting polymer therapeutics to bone.Gene expression profile in osteoclasts from patients with Paget's disease of bone.Transforming Growth Factor β1/Smad4 Signaling Affects Osteoclast Differentiation via Regulation of miR-155 Expression.Bim: guardian of tissue homeostasis and critical regulator of the immune system, tumorigenesis and bone biology.The adaptor protein p62/SQSTM1 in osteoclast signaling pathways.Can platelet-rich plasma (PRP) improve bone healing? A comparison between the theory and experimental outcomes.Mechanisms of spontaneous resolution of acute gouty inflammation.Physiological Bone Remodeling: Systemic Regulation and Growth Factor Involvement.Mathematical Model of Bone Remodeling Captures the Antiresorptive and Anabolic Actions of Various Therapies.Immunology of Gut-Bone Signaling.Molecular signaling mechanisms behind polyphenol-induced bone anabolism.A mechanobiological model of orthodontic tooth movement.Inhibition of osteoclastogenesis by stem cell-derived extracellular matrix through modulation of intracellular reactive oxygen species.Modulation of tumor necrosis factor related apoptosis-inducing ligand (TRAIL) receptors in a human osteoclast model in vitro.Association of SMAD2 polymorphisms with bone mineral density in postmenopausal Korean women.Gap junctional communication is involved in differentiation of osteoclasts from bone marrow and peripheral blood monocytes.

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Transforming growth factor-beta1 (TGF-beta1) induces human osteoclast apoptosis by up-regulating Bim.

http://www.wikidata.org/entity/Q37358112

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on July 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Transforming growth factor-bet ...... poptosis by up-regulating Bim.

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type

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Transforming growth factor-bet ...... poptosis by up-regulating Bim.

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Transforming growth factor-bet ...... poptosis by up-regulating Bim.

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JBC.M109.019372

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Journal of Biological Chemistry

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Transforming growth factor-bet ...... poptosis by up-regulating Bim.

@en

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Estelle Chamoux

http://www.w3.org/2001/XMLSchema#string

Martine Bisson

http://www.w3.org/2001/XMLSchema#string

Nicolas Houde

http://www.w3.org/2001/XMLSchema#string

Sophie Roux

http://www.w3.org/2001/XMLSchema#string

P2860

Transforming growth factor-beta1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3

X-linked inhibitor of apoptosis protein functions as a cofactor in transforming growth factor-beta signaling

Smad transcription factors

Osteopetrosis-like phenotype in latent TGF-beta binding protein 3 deficient mice

Osteoclast differentiation and activation

Pharmacologic inhibition of the TGF-beta type I receptor kinase has anabolic and anti-catabolic effects on bone.

Molecular biology of bone metastasis.

Transforming growth factor beta inhibits formation of osteoclast-like cells in long-term human marrow cultures

TGF-beta receptor-activated p38 MAP kinase mediates Smad-independent TGF-beta responses

Transforming growth factor beta-dependent sequential activation of Smad, Bim, and caspase-9 mediates physiological apoptosis in gastric epithelial cells.

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23397-23404

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scholarly article

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10.1074/JBC.M109.019372

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35

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284

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2009-07-01T00:00:00Z

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26337728

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19574221

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2749113

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