Beta-adrenergic stimulation and myocardial function in the failing heart.
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Counteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein PhosphatasesPKA phosphorylation of the small heat-shock protein Hsp20 enhances its cardioprotective effectsThe relationship between the MMP system, adrenoceptors and phosphoprotein phosphatasesSimilarly potent inhibition of adenylyl cyclase by P-site inhibitors in hearts from wild type and AC5 knockout miceSmall molecule AKAP-protein kinase A (PKA) interaction disruptors that activate PKA interfere with compartmentalized cAMP signaling in cardiac myocytescGMP Signals Modulate cAMP Levels in a Compartment-Specific Manner to Regulate Catecholamine-Dependent Signaling in Cardiac MyocytesModeling the effects of β1-adrenergic receptor blockers and polymorphisms on cardiac myocyte Ca2+ handling.Recent advances in the discovery of small molecules targeting exchange proteins directly activated by cAMP (EPAC).Basal and β-adrenergic regulation of the cardiac calcium channel CaV1.2 requires phosphorylation of serine 1700Protective Role for LPA3 in Cardiac Hypertrophy Induced by Myocardial Infarction but Not by Isoproterenol.Cardiac troponin T, a sarcomeric AKAP, tethers protein kinase A at the myofilaments.Silencing nox4 in the paraventricular nucleus improves myocardial infarction-induced cardiac dysfunction by attenuating sympathoexcitation and periinfarct apoptosis.{beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.β₂ AR agonists in treatment of chronic heart failure: long path to translation.Integrative modeling of the cardiac ventricular myocyte.Cardiac myocytes and local signaling in nano-domains.Okadaic acid induces matrix metalloproteinase-9 expression in fibroblasts: crosstalk between protein phosphatase inhibition and β-adrenoceptor signallingCardiac Effects of Attenuating Gsα - Dependent SignalingLack of Gαi2 leads to dilative cardiomyopathy and increased mortality in β1-adrenoceptor overexpressing micePharmacological characterization of 1-nitrosocyclohexyl acetate, a long-acting nitroxyl donor that shows vasorelaxant and antiaggregatory effects.MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathy.Divide and conquer: the application of organelle proteomics to heart failure.Targeted gene therapy for the treatment of heart failure.cMyBP-C as a promiscuous substrate: phosphorylation by non-PKA kinases and its potential significance.Gene and cytokine therapy for heart failure: molecular mechanisms in the improvement of cardiac function.A pathway and network review on beta-adrenoceptor signaling and beta blockers in cardiac remodeling.Heart failure-specific changes in protein kinase signalling.Atrial fibrillation and heart failure: cause or effect?I-1-deficiency negatively impacts survival in a cardiomyopathy mouse model.Serine/Threonine Phosphatases in Atrial Fibrillation.Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase.Calcium/Calmodulin-Dependent Protein Kinase II Activity Persists During Chronic β-Adrenoceptor Blockade in Experimental and Human Heart Failure.Contractile Defect Caused by Mutation in MYBPC3 Revealed under Conditions Optimized for Human PSC-Cardiomyocyte FunctionDistinct physiological effects of β1- and β2-adrenoceptors in mouse ventricular myocytes: insights from a compartmentalized mathematical model.Isoproterenol cytotoxicity is dependent on the differentiation state of the cardiomyoblast H9c2 cell line.Mathematical modeling improves EC50 estimations from classical dose-response curves.PPARγ Signaling Mediates the Evolution, Development, Homeostasis, and Repair of the Lung.Constitutively active phosphatase inhibitor-1 improves cardiac contractility in young mice but is deleterious after catecholaminergic stress and with aging.The effect of PKA-mediated phosphorylation of ryanodine receptor on SR Ca2+ leak in ventricular myocytes.Phosphatase-1-inhibitor-1: amplifier or attenuator of catecholaminergic stress?
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Beta-adrenergic stimulation and myocardial function in the failing heart.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Beta-adrenergic stimulation and myocardial function in the failing heart.
@en
Beta-adrenergic stimulation and myocardial function in the failing heart.
@nl
type
label
Beta-adrenergic stimulation and myocardial function in the failing heart.
@en
Beta-adrenergic stimulation and myocardial function in the failing heart.
@nl
prefLabel
Beta-adrenergic stimulation and myocardial function in the failing heart.
@en
Beta-adrenergic stimulation and myocardial function in the failing heart.
@nl
P2860
P1476
Beta-adrenergic stimulation and myocardial function in the failing heart.
@en
P2093
Ali El-Armouche
Thomas Eschenhagen
P2860
P2888
P304
P356
10.1007/S10741-008-9132-8
P577
2009-12-01T00:00:00Z