Estrogen suppresses activation but enhances formation phase of osteogenic response to mechanical stimulation in rat bone.
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Peroral Estradiol Is Sufficient to Induce Carcinogen-Induced Mammary Tumorigenesis in Ovariectomized Rats without ProgesteroneRoBo-1, a novel member of the urokinase plasminogen activator receptor/CD59/Ly-6/snake toxin family selectively expressed in rat bone and growth plate cartilageEstrogen enhances mechanical stress-induced prostaglandin production by bone cells from elderly women.Pathogenesis of age-related osteoporosis: impaired mechano-responsiveness of bone is not the culprit.The Contribution of Experimental in vivo Models to Understanding the Mechanisms of Adaptation to Mechanical Loading in BoneSex steroid actions in male boneMechanical loading-related bone gain is enhanced by tamoxifen but unaffected by fulvestrant in female miceThe impact of low-magnitude high-frequency vibration on fracture healing is profoundly influenced by the oestrogen status in mice.Estrogen augments shear stress-induced signaling and gene expression in osteoblast-like cells via estrogen receptor-mediated expression of beta1-integrin.Functional adaptation in female rats: the role of estrogen signalingA cross-sectional study of the relationship between cortical bone and high-impact activity in young adult males and females.Estrogen receptor-α is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2.FSH and TSH in the regulation of bone mass: the pituitary/immune/bone axis.Vascular pedicle ossification of free fibular flap: is it a rare phenomenon? Is it possible to avoid this risk?Low-magnitude high-frequency vibration enhances gene expression related to callus formation, mineralization and remodeling during osteoporotic fracture healing in rats.Estrogen regulates stemness and senescence of bone marrow stromal cells to prevent osteoporosis via ERβ-SATB2 pathway.Trabecular bone response to mechanical and parathyroid hormone stimulation: the role of mechanical microenvironment.[Mechanobiology and bone metabolism: Clinical relevance for fracture treatment].Mechano-transduction in osteoblastic cells involves strain-regulated estrogen receptor alpha-mediated control of insulin-like growth factor (IGF) I receptor sensitivity to Ambient IGF, leading to phosphatidylinositol 3-kinase/AKT-dependent Wnt/LRP5
P2860
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P2860
Estrogen suppresses activation but enhances formation phase of osteogenic response to mechanical stimulation in rat bone.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
Estrogen suppresses activation ...... nical stimulation in rat bone.
@en
Estrogen suppresses activation ...... nical stimulation in rat bone.
@nl
type
label
Estrogen suppresses activation ...... nical stimulation in rat bone.
@en
Estrogen suppresses activation ...... nical stimulation in rat bone.
@nl
prefLabel
Estrogen suppresses activation ...... nical stimulation in rat bone.
@en
Estrogen suppresses activation ...... nical stimulation in rat bone.
@nl
P2093
P2860
P356
P1476
Estrogen suppresses activation ...... nical stimulation in rat bone.
@en
P2093
C J Jagger
T J Chambers
P2860
P304
P356
10.1172/JCI119047
P407
P577
1996-11-01T00:00:00Z