Estrogen receptor-α is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2.
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The role of estrogen and androgen receptors in bone health and diseasePhysiological mechanisms and therapeutic potential of bone mechanosensing.Effects of sex steroids on bones and muscles: Similarities, parallels, and putative interactions in health and diseaseLocal mechanical stimuli regulate bone formation and resorption in mice at the tissue level.Regulation of early growth response 2 expression by secreted frizzled related protein 1Retrieved bone-anchored percutaneous amputation prosthesis showing maintained osseointegration after 11 years-a case reportEffect of GH/IGF-1 on Bone Metabolism and Osteoporsosis.Protein kinase Cα (PKCα) regulates bone architecture and osteoblast activity.The Contribution of Experimental in vivo Models to Understanding the Mechanisms of Adaptation to Mechanical Loading in BoneIncrease of Compact Bone Thickness in Rat Tibia after Implanting MgO into the Bone Marrow Cavity.Sex steroid actions in male boneEstrogens and Androgens in Skeletal Physiology and Pathophysiology.Age-related impairment of bones' adaptive response to loading in mice is associated with sex-related deficiencies in osteoblasts but no change in osteocytes.Minireview: nuclear receptor regulation of osteoclast and bone remodeling.Planar cell polarity aligns osteoblast division in response to substrate strainWnt16 Is Associated with Age-Related Bone Loss and Estrogen Withdrawal in Murine Bone.Estrogen receptor-α signaling in osteoblast progenitors stimulates cortical bone accrualEstrogen Receptor α Regulates Dlx3-Mediated Osteoblast Differentiation.Estrogen receptor-α in osteocytes is important for trabecular bone formation in male miceEstrogen synthesis and signaling pathways during aging: from periphery to brainEstrogen receptor α mediates proliferation of osteoblastic cells stimulated by estrogen and mechanical strain, but their acute down-regulation of the Wnt antagonist Sost is mediated by estrogen receptor β.SERMs have substance-specific effects on bone, and these effects are mediated via ERαAF-1 in female mice.Estrogen receptors' roles in the control of mechanically adaptive bone (re)modelingThe molecular basis of bone mechanotransduction.Compressive loading of the murine tibia reveals site-specific micro-scale differences in adaptation and maturation rates of bone.Bone corticalization requires local SOCS3 activity and is promoted by androgen action via interleukin-6.Mouse models to evaluate the role of estrogen receptor α in skeletal maintenance and adaptation.Liver-derived IGF-I regulates cortical bone mass but is dispensable for the osteogenic response to mechanical loading in female mice.Osteoprotective Effects of Estrogen in the Maxillary Bone Depend on ERα.[Mechanobiology and bone metabolism: Clinical relevance for fracture treatment].
P2860
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P2860
Estrogen receptor-α is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@ast
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@en
type
label
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@ast
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@en
prefLabel
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@ast
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@en
P2093
P2860
P50
P356
P1476
Estrogen receptor-α is require ...... tivation function 1 but not 2.
@en
P2093
Andrée Krust
Baruch Frenkel
Joanna S Price
Lance E Lanyon
Leanne Saxon
Maria C Antal
Petra Henning
P2860
P304
P356
10.1002/JBMR.1754
P50
P577
2013-02-01T00:00:00Z