CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
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Fas-ligand: privilege and perilHigh-risk corneal allografts: A therapeutic challengeOcular Immune Privilege and TransplantationProlonging survival of corneal transplantation by selective sphingosine-1-phosphate receptor 1 agonistHepatocyte nuclear factor (HNF)-4alpha induces expression of endothelial Fas ligand (FasL) to prevent cancer cell transmigration: a novel defense mechanism of endothelium against cancer metastasis.Natural Killer T Cells Contribute to Neutrophil Recruitment and Ocular Tissue Damage in a Model of Intraocular Tumor RejectionSoluble Fas and Fas ligand in human tear fluid after photorefractive keratectomyEgr family members regulate nonlymphoid expression of Fas ligand, TRAIL, and tumor necrosis factor during immune responsesCorneal graft rejection occurs despite Fas ligand expression and apoptosis of infiltrating cells.Many checkpoints on the road to cell death: regulation of Fas-FasL interactions and Fas signaling in peripheral immune responses.Pathogenesis of onchocercal keratitis (River blindness).Feasibility study of lamellar keratoplasty in a murine model.Gene therapy in transplantation.Application of a Fas ligand encoding a recombinant adenovirus vector for prolongation of transgene expression.Involvement of Fas-Fas ligand interactions in graft rejection.Apoptosis and CD95 ligand in immune privileged sites.Opposing roles for membrane bound and soluble Fas ligand in glaucoma-associated retinal ganglion cell death.Targeting immune privilege to prevent pathogenic neovascularizationExpression of Fas (CD95/APO-1) ligand by human breast cancers: significance for tumor immune privilegeGene delivery to the corneal endothelium.Autoimmune thyroid disease: new models of cell death in autoimmunity.Corneal transplantation and immune privilege.Keratocyte apoptosis and failure of corneal allograftsGalectin-9-mediated protection from allo-specific T cells as a mechanism of immune privilege of corneal allografts.Loss of the perforin cytotoxic pathway predisposes mice to experimental cytomegalovirus retinitis.Systemic immunological tolerance to ocular antigens is mediated by TRAIL-expressing CD8+ T cells.Age-dependent changes in FasL (CD95L) modulate macrophage function in a model of age-related macular degenerationEffect of reactive oxygen species generation in rabbit corneal epithelial cells on inflammatory and apoptotic signaling pathways in the presence of high osmotic pressure.Armed response: how dying cells influence T-cell functions.Anterior chamber associated immune deviation (ACAID): regulation, biological relevance, and implications for therapy.Cell death and immune privilege.Immunology and immunomodulation of corneal transplantation.Recent developments in the pharmacological treatment and prevention of corneal graft rejection.The potential of antibody-based immunosuppressive agents for corneal transplantation.Murine corneal transplantation: a model to study the most common form of solid organ transplantation.The immune privilege of corneal grafts.Cell subpopulations in failed human corneal grafts.Apoptosis is a prominent feature of acute anterior uveitis in the Fischer 344 rat.Expression of Fas ligand by human gastric adenocarcinomas: a potential mechanism of immune escape in stomach cancerManagement of high-risk corneal grafts.
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CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
@en
CD95 ligand
@nl
type
label
CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
@en
CD95 ligand
@nl
prefLabel
CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
@en
CD95 ligand
@nl
P2093
P2860
P356
P1476
CD95 ligand (FasL)-induced apoptosis is necessary for corneal allograft survival.
@en
P2093
P2860
P304
P356
10.1172/JCI119173
P407
P577
1997-02-01T00:00:00Z