Immunodesign of experimental sepsis by cecal ligation and puncture.
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Nutritional and Nanotechnological Modulators of MicrogliaCD1d- and MR1-Restricted T Cells in SepsisG-protein-coupled receptor kinases in inflammation and diseaseT-cell-mediated immunity and the role of TRAIL in sepsis-induced immunosuppressionThe inflammatory response in sepsisLung Transcriptomics during Protective Ventilatory Support in Sepsis-Induced Acute Lung InjuryInflammation-induced acute phase response in skeletal muscle and critical illness myopathyFactor XI Deficiency Alters the Cytokine Response and Activation of Contact Proteases during Polymicrobial Sepsis in MiceBeneficial Effects of Anti-Interleukin-6 Antibodies on Impaired Gastrointestinal Motility, Inflammation and Increased Colonic Permeability in a Murine Model of Sepsis Are Most Pronounced When Administered in a Preventive SetupFoxp3+ regulatory T cells are required for recovery from severe sepsisImmunosuppression after sepsis: systemic inflammation and sepsis induce a loss of naïve T-cells but no enduring cell-autonomous defects in T-cell functionPharmacological inhibitors of TRPV4 channels reduce cytokine production, restore endothelial function and increase survival in septic miceSuppression of inflammation by a synthetic histone mimicRegulation of drug-metabolizing enzymes by local and systemic liver injuriesPlatelets induce apoptosis during sepsis in a contact-dependent manner that is inhibited by GPIIb/IIIa blockadeEndothelial cell-derived CD95 ligand serves as a chemokine in induction of neutrophil slow rolling and adhesionChemical chaperone TUDCA prevents apoptosis and improves survival during polymicrobial sepsis in miceAnimal models of sepsis and sepsis-induced kidney injuryUropod elongation is a common final step in leukocyte extravasation through inflamed vessels.Sepsis otopathy: experimental sepsis leads to significant hearing impairment due to apoptosis and glutamate excitotoxicity in murine cochleaLy6Chigh Monocytes Protect against Kidney Damage during Sepsis via a CX3CR1-Dependent Adhesion MechanismDetrimental effect of apoptosis of lymphocytes at an early time point of experimental abdominal sepsis.C5a regulates IL-12+ DC migration to induce pathogenic Th1 and Th17 cells in sepsis.Resolvin D2 is a potent regulator of leukocytes and controls microbial sepsisMyocardial oxidative stress correlates with left ventricular dysfunction on strain echocardiography in a rodent model of sepsis.Inositol polyphosphate multikinase promotes Toll-like receptor-induced inflammation by stabilizing TRAF6.Protectin DX increases survival in a mouse model of sepsis by ameliorating inflammation and modulating macrophage phenotype.Apoptotic adipose-derived mesenchymal stem cell therapy protects against lung and kidney injury in sepsis syndrome caused by cecal ligation puncture in rats.Intravenous Arginine Administration Promotes Proangiogenic Cells Mobilization and Attenuates Lung Injury in Mice with Polymicrobial SepsisKinetics and protective role of autophagy in a mouse cecal ligation and puncture-induced sepsisCritical illness induces nutrient-independent adipogenesis and accumulation of alternatively activated tissue macrophagesAtorvastatin improves survival in septic rats: effect on tissue inflammatory pathway and on insulin signaling.Recombinant human plasma phospholipid transfer protein (PLTP) to prevent bacterial growth and to treat sepsis.The ubiquitin ligase ZNRF1 promotes caveolin-1 ubiquitination and degradation to modulate inflammation.Surviving murine experimental sepsis affects the function and morphology of the inner ear.Disrupting ceramide-CD300f interaction prevents septic peritonitis by stimulating neutrophil recruitment.Combined administration of a sedative dose sevoflurane and 60% oxygen reduces inflammatory responses to sepsis in animals and in human PMBCs.Amelioration of sepsis by inhibiting sialidase-mediated disruption of the CD24-SiglecG interaction.Effect of siRNA against NF-κB on sepsis‑induced acute lung injury in a mouse model.Trichinella spiralis excretory-secretory products protect against polymicrobial sepsis by suppressing MyD88 via mannose receptor.
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Immunodesign of experimental sepsis by cecal ligation and puncture.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Immunodesign of experimental sepsis by cecal ligation and puncture.
@en
Immunodesign of experimental sepsis by cecal ligation and puncture.
@nl
type
label
Immunodesign of experimental sepsis by cecal ligation and puncture.
@en
Immunodesign of experimental sepsis by cecal ligation and puncture.
@nl
prefLabel
Immunodesign of experimental sepsis by cecal ligation and puncture.
@en
Immunodesign of experimental sepsis by cecal ligation and puncture.
@nl
P2860
P50
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P1476
Immunodesign of experimental sepsis by cecal ligation and puncture.
@en
P2093
Michael A Flierl
P2860
P2888
P356
10.1038/NPROT.2008.214
P577
2009-01-01T00:00:00Z
P5875
P6179
1036382062