Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
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The central GLP-1: implications for food and drug rewardWe are ageingExendin-4 differentiation of a human pancreatic duct cell line into endocrine cells: involvement of PDX-1 and HNF3beta transcription factorsThe L-Cell in Nutritional Sensing and the Regulation of AppetiteNovel GLP-1 fusion chimera as potent long acting GLP-1 receptor agonistRole of vesicular monoamine transporter type 2 in rodent insulin secretion and glucose metabolism revealed by its specific antagonist tetrabenazineNifedipine suppresses neointimal thickening by its inhibitory effect on vascular smooth muscle cell growth via a MEK-ERK pathway coupling with Pyk2Expression of glucagon-like peptide-1 (GLP-1) receptor and the effect of GLP-1-(7-36) amide on insulin release by pancreatic islets during rat ontogenic development.The human glucagon-like peptide-1 analogue liraglutide preserves pancreatic beta cells via regulation of cell kinetics and suppression of oxidative and endoplasmic reticulum stress in a mouse model of diabetes.The impact of dietary energy intake on cognitive aging.Exendin-4 improves blood glucose control in both young and aging normal non-diabetic mice, possible contribution of beta cell independent effects.Age-related impairment of pancreatic Beta-cell function: pathophysiological and cellular mechanisms.Incretin mimetics as pharmacologic tools to elucidate and as a new drug strategy to treat traumatic brain injuryEffect of cyclosporine A on hepatic carbohydrate metabolism and hepatic gene expression in rat.Diminished mTOR signaling: a common mode of action for endocrine longevity factors.Liraglutide increases FGF-21 activity and insulin sensitivity in high fat diet and adiponectin knockdown induced insulin resistance.Therapeutic potential of dipeptidyl peptidase IV inhibitors for the treatment of type 2 diabetes.Therapeutic Approaches for Preserving or Restoring Pancreatic β-Cell Function and Mass.Bidirectional metabolic regulation of neurocognitive function.Mice lacking dipeptidyl peptidase IV are protected against obesity and insulin resistanceThe role of gut hormones in glucose homeostasisExendin-4 increases islet amyloid deposition but offsets the resultant beta cell toxicity in human islet amyloid polypeptide transgenic mouse islets.Evidence of GLP-1-mediated neuroprotection in an animal model of pyridoxine-induced peripheral sensory neuropathy.Glucagon-like peptide 1: evolution of an incretin into a treatment for diabetes.Blockade of cannabinoid 1 receptor improves GLP-1R mediated insulin secretion in mice.Therapy in the early stage: incretins.Targeting beta-cell mass in type 2 diabetes: promise and limitations of new drugs based on incretinsClinical review: The extrapancreatic effects of glucagon-like peptide-1 and related peptidesTolerance, fermentation, and cytokine expression in healthy aged male C57BL/6J mice fed resistant starch.Dietary resistant starch improves selected brain and behavioral functions in adult and aged rodents.Possible role of GLP-1 and its agonists in the treatment of type 1 diabetes mellitus.Discovery and development of exenatide: the first antidiabetic agent to leverage the multiple benefits of the incretin hormone, GLP-1.The place of GLP-1-based therapy in diabetes management: differences between DPP-4 inhibitors and GLP-1 receptor agonists.Pharmacological profile, efficacy and safety of lixisenatide in type 2 diabetes mellitus.GLP-1-mediated gene therapy approaches for diabetes treatment.Oral 2-oleyl glyceryl ether improves glucose tolerance in mice through the GPR119 receptor.How Does Exercise Reduce the Rate of Age-Associated Cognitive Decline? A Review of Potential Mechanisms.GLP-1 receptor independent pathways: emerging beneficial effects of GLP-1 breakdown products.Insulin promoter factor-1 gene mutation linked to early-onset type 2 diabetes mellitus directs expression of a dominant negative isoprotein.Increased secretion and expression of amylin in spontaneously diabetic Goto-Kakizaki rats treated with rhGLP-1 (7-36).
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Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on June 1997
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
@en
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats.
@nl
type
label
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
@en
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats.
@nl
prefLabel
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
@en
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats.
@nl
P2093
P2860
P356
P1476
Glucagon-like peptide-1 can reverse the age-related decline in glucose tolerance in rats
@en
P2093
Holloway HW
Montrose-Rafizadeh C
Perfetti R
P2860
P304
P356
10.1172/JCI119482
P407
P577
1997-06-01T00:00:00Z