The critical early proinflammatory events associated with idiopathic pneumonia syndrome in irradiated murine allogeneic recipients are due to donor T cell infusion and potentiated by cyclophosphamide
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Leukocyte migration and graft-versus-host disease.Idiopathic Pneumonia Syndrome and Thrombotic Microangiopathy Following Nonmyeloablative Haploidentical Peripheral Blood Stem Cell Transplantation and Posttransplant Cyclophosphamide: A Case ReportMechanisms of lung injury after bone marrow transplantation.Influence of oral beclomethasone dipropionate on early non-infectious pulmonary outcomes after allogeneic hematopoietic cell transplantation: results from two randomized trials.Lung parenchyma-derived IL-6 promotes IL-17A-dependent acute lung injury after allogeneic stem cell transplantationDifferential roles of IL-1 and TNF-alpha on graft-versus-host disease and graft versus leukemia.Surfactant Protein A: regulation of innate and adaptive immune responses in lung inflammation.Pairwise protein expression classifier for candidate biomarker discovery for early detection of human disease prognosis.The impact of soluble tumor necrosis factor receptor etanercept on the treatment of idiopathic pneumonia syndrome after allogeneic hematopoietic stem cell transplantation.Diffuse alveolar hemorrhage in hematopoietic stem cell transplant recipients.TNF-receptor inhibitor therapy for the treatment of children with idiopathic pneumonia syndrome. A joint Pediatric Blood and Marrow Transplant Consortium and Children's Oncology Group Study (ASCT0521).Translational research efforts in biomarkers and biology of early transplant-related complications.Evidence for early fibrosis and increased airway resistance in bone marrow transplant recipient mice deficient in MMP12.Prevention of GVHD while sparing GVL effect by targeting Th1 and Th17 transcription factor T-bet and RORγt in mice.An official American Thoracic Society research statement: noninfectious lung injury after hematopoietic stem cell transplantation: idiopathic pneumonia syndrome.Pulmonary complications of solid organ and hematopoietic stem cell transplantation.Rust and corrosion in hematopoietic stem cell transplantation: the problem of iron and oxidative stress.Acute lung injury after allogeneic stem cell transplantation: is the lung a target of acute graft-versus-host disease?A new murine model for bronchiolitis obliterans post-bone marrow transplant.Decreased asbestos-induced lung inflammation and fibrosis after radiation and bone marrow transplant.A role for TNF receptor type II in leukocyte infiltration into the lung during experimental idiopathic pneumonia syndrome.Simultaneous absence of surfactant proteins A and D increases lung inflammation and injury after allogeneic HSCT in mice.Limited restoration of cystic fibrosis lung epithelium in vivo with adult bone marrow-derived cellsA Critical Care and Transplantation-Based Approach to Acute Respiratory Failure after Hematopoietic Stem Cell Transplantation in Children.Club cell secretory protein improves survival in a murine obliterative bronchiolitis model.Pleiotropic effects of transforming growth factor-β in hematopoietic stem-cell transplantation.A survey of changing trends in modelling radiation lung injury in mice: bringing out the good, the bad, and the uncertain.Targeting the Canonical Nuclear Factor-κB Pathway with a High-Potency IKK2 Inhibitor Improves Outcomes in a Mouse Model of Idiopathic Pneumonia Syndrome.The association between platelet transfusion and idiopathic pneumonia syndrome is unaffected by platelet product type.Surfactant protein A decreases lung injury and mortality after murine marrow transplantation.Human surfactant protein a suppresses T cell-dependent inflammation and attenuates the manifestations of idiopathic pneumonia syndrome in mice.Effects of oxidant stress on inflammation and survival of iNOS knockout mice after marrow transplantation.Idiopathic pneumonia after marrow transplantation: when are antioxidants effective?Idiopathic pneumonia syndrome after syngeneic bone marrow transplant in mice.Post-BMT lung injury occurs independently of the expression of CCL2 or its receptor, CCR2, on host cells.Absence of host tumor necrosis factor receptor 1 attenuates manifestations of idiopathic pneumonia syndrome.Myeloperoxidase deficiency enhances inflammation after allogeneic marrow transplantation.Surfactant protein A is a required mediator of keratinocyte growth factor after experimental marrow transplantation.Early onset of acute immune-mediated lung injury in a child undergoing allogeneic peripheral blood transplantation.
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P2860
The critical early proinflammatory events associated with idiopathic pneumonia syndrome in irradiated murine allogeneic recipients are due to donor T cell infusion and potentiated by cyclophosphamide
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
The critical early proinflamma ...... otentiated by cyclophosphamide
@en
The critical early proinflamma ...... tentiated by cyclophosphamide.
@nl
type
label
The critical early proinflamma ...... otentiated by cyclophosphamide
@en
The critical early proinflamma ...... tentiated by cyclophosphamide.
@nl
prefLabel
The critical early proinflamma ...... otentiated by cyclophosphamide
@en
The critical early proinflamma ...... tentiated by cyclophosphamide.
@nl
P2093
P2860
P356
P1476
The critical early proinflamma ...... otentiated by cyclophosphamide
@en
P2093
A Panoskaltsis-Mortari
B R Blazar
D A Vallera
D H Ingbar
O D Wangensteen
P A Taylor
T M Yaeger
P2860
P304
P356
10.1172/JCI119612
P407
P577
1997-09-01T00:00:00Z