Lipopolysaccharide-stimulated osteoclastogenesis is mediated by tumor necrosis factor via its P55 receptor.
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Role of the A20-TRAF6 axis in lipopolysaccharide-mediated osteoclastogenesisOsteoclasts: what do they do and how do they do it?The role of TNF-receptor family members and other TRAF-dependent receptors in bone resorption.Bone loss. Therapeutic approaches for preventing bone loss in inflammatory arthritisAttenuation of bone mass and increase of osteoclast formation in decoy receptor 3 transgenic mice.An extensive phenotypic characterization of the hTNFalpha transgenic mice.In vitro evaluation of the effects of low-intensity Nd:YAG laser irradiation on the inflammatory reaction elicited by bacterial lipopolysaccharide adherent to titanium dental implants.Ethyl acetate fraction from Cudrania tricuspidata inhibits IL-1beta-stimulated osteoclast differentiation through downregulation of MAPKs, c-Fos and NFATc1Lipopolysaccharide differentially affects the osteogenic differentiation of periodontal ligament stem cells and bone marrow mesenchymal stem cells through Toll-like receptor 4 mediated nuclear factor κB pathwayTumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interactionCloning and characterization of osteoclast precursors from the RAW264.7 cell lineContradictory Role of CD97 in Basal and Tumor Necrosis Factor-Induced Osteoclastogenesis In Vivo.Mice lacking beta3 integrins are osteosclerotic because of dysfunctional osteoclastsLeptin potentiates Prevotella intermedia lipopolysaccharide-induced production of TNF-alpha in monocyte-derived macrophages.Interleukin-1 and tumor necrosis factor activities partially account for calvarial bone resorption induced by local injection of lipopolysaccharide.Impaired bone resorption by lipopolysaccharide in vivo in mice deficient in the prostaglandin E receptor EP4 subtype.Regulation of sealing ring formation by L-plastin and cortactin in osteoclasts.Pathological fractures in paediatric patients with inflammatory bowel disease.Osteoprotegerin is a receptor for the cytotoxic ligand TRAIL.Tumor necrosis factor-α signaling in macrophages.Osteoclasts; culprits in inflammatory osteolysis.Tumour necrosis factor superfamily cytokines and the pathogenesis of inflammatory osteolysisAryl hydrocarbon receptors in osteoclast lineage cells are a negative regulator of bone mass.Skeletal health of children and adolescents with inflammatory bowel disease.Correlating RANK ligand/RANK binding kinetics with osteoclast formation and functionIL-12 Inhibits Lipopolysaccharide Stimulated Osteoclastogenesis in Mice.Rhinacanthin C Inhibits Osteoclast Differentiation and Bone Resorption: Roles of TRAF6/TAK1/MAPKs/NF-κB/NFATc1 SignalingTumor necrosis factor-alpha mediates orthopedic implant osteolysis.Interleukin-1 and tumor necrosis factor receptor signaling is not required for bacteria-induced osteoclastogenesis and bone loss but is essential for protecting the host from a mixed anaerobic infection.Macrophage glucocorticoid receptors regulate Toll-like receptor 4-mediated inflammatory responses by selective inhibition of p38 MAP kinaseImpediment of NEMO oligomerization inhibits osteoclastogenesis and osteolysisOsteoprotegerin Regulates Pancreatic β-Cell Homeostasis upon Microbial InvasionThe role of SH3BP2 in the pathophysiology of cherubism.Complications of mastoiditis in children at the onset of a new millennium.Cannabinoids and bone: endocannabinoids modulate human osteoclast function in vitroExcavatolide B Attenuates Rheumatoid Arthritis through the Inhibition of Osteoclastogenesis.An essential role of cytosolic phospholipase A2alpha in prostaglandin E2-mediated bone resorption associated with inflammationThe IkappaB function of NF-kappaB2 p100 controls stimulated osteoclastogenesisMacrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss.Estrogen decreases TNF gene expression by blocking JNK activity and the resulting production of c-Jun and JunD.
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Lipopolysaccharide-stimulated osteoclastogenesis is mediated by tumor necrosis factor via its P55 receptor.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@en
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@nl
type
label
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@en
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@nl
prefLabel
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@en
Lipopolysaccharide-stimulated ...... s factor via its P55 receptor.
@nl
P2093
P2860
P356
P1476
Lipopolysaccharide-stimulated ...... is factor via its P55 receptor
@en
P2093
P2860
P304
P356
10.1172/JCI119679
P407
P577
1997-09-01T00:00:00Z