Restoration of NET formation by gene therapy in CGD controls aspergillosis. - Wikidata - wikimedia - TriplyDB

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

http://www.wikidata.org/entity/Q37372812

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Neutrophil extracellular traps contain calprotectin, a cytosolic protein complex involved in host defense against Candida albicans Production of extracellular traps against Aspergillus fumigatus in vitro and in infected lung tissue is dependent on invading neutrophils and influenced by hydrophobin RodA Rab27a is essential for the formation of neutrophil extracellular traps (NETs) in neutrophil-like differentiated HL60 cells Impairment of neutrophil extracellular trap degradation is associated with lupus nephritis New Insights into Neutrophil Extracellular Traps: Mechanisms of Formation and Role in Inflammation Platelets in Pulmonary Immune Responses and Inflammatory Lung Diseases Advances of gene therapy for primary immunodeficiencies First Line of Defense: Innate Cell-Mediated Control of Pulmonary Aspergillosis Do neutrophil extracellular traps contribute to the heightened risk of thrombosis in inflammatory diseases?Sphingolipids as Regulators of the Phagocytic Response to Fungal Infections Extracellular DNA traps in allergic, infectious, and autoimmune diseases Miraculous catch by neutrophils NETs Gene therapy for primary immunodeficiencies Peptidylarginine deiminases in citrullination, gene regulation, health and pathogenesis Crossroads of coagulation and innate immunity: the case of deep vein thrombosis Chronic granulomatous disease Mendelian genetics of human susceptibility to fungal infection Classical ROS-dependent and early/rapid ROS-independent release of Neutrophil Extracellular Traps triggered by Leishmania parasites.Protection of Candida parapsilosis from neutrophil killing through internalization by human endothelial cells Trypanosoma cruzi and Its Soluble Antigens Induce NET Release by Stimulating Toll-Like Receptors Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis Social amoebae trap and kill bacteria by casting DNA nets Modulation of neutrophil NETosis: interplay between infectious agents and underlying host physiology.Oxidative stress in the pathogenesis of atherothrombosis associated with anti-phospholipid syndrome and systemic lupus erythematosus: new therapeutic approaches NETosis as Source of Autoantigens in Rheumatoid Arthritis Neutrophils: Between host defence, immune modulation, and tissue injury The immune interplay between the host and the pathogen in Aspergillus fumigatus lung infection Automatic quantification of in vitro NET formation Loss of mitochondrial protein Fus1 augments host resistance to Acinetobacter baumannii infection Influence of neutrophil defects on Burkholderia cepacia complex pathogenesis.Live imaging of disseminated candidiasis in zebrafish reveals role of phagocyte oxidase in limiting filamentous growth.ETosis: A Microbicidal Mechanism beyond Cell Death.Neutrophil extracellular traps: is immunity the second function of chromatin?Monocyte- and macrophage-targeted NADPH oxidase mediates antifungal host defense and regulation of acute inflammation in mice.Neutrophils sense microbe size and selectively release neutrophil extracellular traps in response to large pathogens.Elevated homocysteine levels in type 2 diabetes induce constitutive neutrophil extracellular traps.Neutrophil Extracellular Traps of Cynoglossus semilaevis: Production Characteristics and Antibacterial Effect.Retrovirus gene therapy for X-linked chronic granulomatous disease can achieve stable long-term correction of oxidase activity in peripheral blood neutrophils.Gene therapy for PIDs: progress, pitfalls and prospects NADPH oxidase limits innate immune responses in the lungs in mice

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Restoration of NET formation by gene therapy in CGD controls aspergillosis.

http://www.wikidata.org/entity/Q37372812

description

article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 18 June 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@en

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@nl

type

label

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@en

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@nl

prefLabel

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@en

Restoration of NET formation by gene therapy in CGD controls aspergillosis.

@nl

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BLOOD-2009-05-221606

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Restoration of NET formation by gene therapy in CGD controls aspergillosis.

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Abdul Hakkim

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Arturo Zychlinsky

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Reinhard A Seger

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Ulrich Siler

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Volker Brinkmann

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P2860

How neutrophils kill microbes

Neutrophil extracellular traps kill bacteria

Insertional oncogenesis in 4 patients after retrovirus-mediated gene therapy of SCID-X1

Neutrophil extracellular traps capture and kill Candida albicans yeast and hyphal forms

Novel cell death program leads to neutrophil extracellular traps

Aspergillus fumigatus and aspergillosis

Chronic granulomatous disease. Report on a national registry of 368 patients.

Interaction of Aspergillus fumigatus Spores with Human Leukocytes and Serum.

Correction of X-linked chronic granulomatous disease by gene therapy, augmented by insertional activation of MDS1-EVI1, PRDM16 or SETBP1.

Killing of Aspergillus fumigatus by alveolar macrophages is mediated by reactive oxidant intermediates.

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Janine Reichenbach

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