Regulated overexpression of interleukin 11 in the lung. Use to dissociate development-dependent and -independent phenotypes
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Animal Models, Learning Lessons to Prevent and Treat Neonatal Chronic Lung DiseaseThe construction of transgenic and gene knockout/knockin mouse models of human diseaseReduced tumor necrosis factor-alpha and transforming growth factor-beta1 expression in the lungs of inbred mice that fail to develop fibroproliferative lesions consequent to asbestos exposureMetabolic consequences of adenosine deaminase deficiency in mice are associated with defects in alveogenesis, pulmonary inflammation, and airway obstructionRequirement of transcription factor NFAT in developing atrial myocardiumBcl-2-related protein A1 is an endogenous and cytokine-stimulated mediator of cytoprotection in hyperoxic acute lung injury.Animal models for chronic obstructive pulmonary disease: age of klotho and marlboro mice.Lymphotoxin-alpha contributes to lymphangiogenesis.Nuclear hormone retinoid X receptor (RXR) negatively regulates the glucose-stimulated insulin secretion of pancreatic ß-cellsAnimal models of chronic obstructive pulmonary disease.Inducible targeting of IL-13 to the adult lung causes matrix metalloproteinase- and cathepsin-dependent emphysemaAdenosine metabolism and murine strain-specific IL-4-induced inflammation, emphysema, and fibrosis.Mesenchymal-epithelial interactions in lung development and repair: are modeling and remodeling the same process?Chronic obstructive pulmonary disease * 3: Experimental animal models of pulmonary emphysema.Inducible expression of keratinocyte growth factor (KGF) in mice inhibits lung epithelial cell death induced by hyperoxia.Epithelial cell modulation of airway fibrosis in asthma.Selective p38α mitogen-activated protein kinase inhibitor attenuates lung inflammation and fibrosis in IL-13 transgenic mouse model of asthma.Regulated angiogenesis and vascular regression in mice overexpressing vascular endothelial growth factor in airways.Impaired mesenchymal cell function in Gata4 mutant mice leads to diaphragmatic hernias and primary lung defects.Emphysematous lesions, inflammation, and fibrosis in the lungs of transgenic mice overexpressing platelet-derived growth factor.IL-18 induces emphysema and airway and vascular remodeling via IFN-γ, IL-17A, and IL-13Building better mouse models of asthma.Vascular endothelial growth factor (VEGF) induces remodeling and enhances TH2-mediated sensitization and inflammation in the lung.Interferon gamma induction of pulmonary emphysema in the adult murine lungActivated Akt protects the lung from oxidant-induced injury and delays death of mice.Essential role of nuclear factor kappaB in the induction of eosinophilia in allergic airway inflammationDivergent roles of endothelial NF-kappaB in multiple organ injury and bacterial clearance in mouse models of sepsis.Developmental regulation of NO-mediated VEGF-induced effects in the lung.Expression of the reverse tetracycline-transactivator gene causes emphysema-like changes in mice.Elastase- and LPS-exposed mice display altered responses to rhinovirus infection.Determinants of initiation and progression of idiopathic pulmonary fibrosis.Nuclear factor of activated T cells and serum response factor cooperatively regulate the activity of an alpha-actin intronic enhancer.Overlapping and enzyme-specific contributions of matrix metalloproteinases-9 and -12 in IL-13-induced inflammation and remodeling.Pulmonary overexpression of IL-9 induces Th2 cytokine expression, leading to immune pathologyTargeted lung expression of interleukin-11 enhances murine tolerance of 100% oxygen and diminishes hyperoxia-induced DNA fragmentationIL-13 stimulates vascular endothelial cell growth factor and protects against hyperoxic acute lung injury.Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production.Inducible lung-specific expression of RANTES: preferential recruitment of neutrophils.Modulation of cell growth and transformation by doxycycline-regulated FGF-2 expression in NIH-3T3 cells.Epithelial reticulon 4B (Nogo-B) is an endogenous regulator of Th2-driven lung inflammation.
P2860
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P2860
Regulated overexpression of interleukin 11 in the lung. Use to dissociate development-dependent and -independent phenotypes
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Regulated overexpression of in ...... nt and -independent phenotypes
@en
Regulated overexpression of in ...... t and -independent phenotypes.
@nl
type
label
Regulated overexpression of in ...... nt and -independent phenotypes
@en
Regulated overexpression of in ...... t and -independent phenotypes.
@nl
prefLabel
Regulated overexpression of in ...... nt and -independent phenotypes
@en
Regulated overexpression of in ...... t and -independent phenotypes.
@nl
P2093
P2860
P356
P1476
Regulated overexpression of in ...... nt and -independent phenotypes
@en
P2093
P2860
P304
P356
10.1172/JCI119792
P407
P577
1997-11-01T00:00:00Z