Intercellular communication of cellular stress monitored by gamma-H2AX induction. - Wikidata - wikimedia - TriplyDB

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

http://www.wikidata.org/entity/Q37376590

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Evaluating biomarkers to model cancer risk post cosmic ray exposure The P5 disulfide switch: taming the aging unfolded protein response Preventive or potential therapeutic value of nutraceuticals against ionizing radiation-induced oxidative stress in exposed subjects and frequent fliers Low doses of gamma-irradiation induce an early bystander effect in zebrafish cells which is sufficient to radioprotect cells Oxidative DNA damage caused by inflammation may link to stress-induced non-targeted effects.Epigenetic control of mobile DNA as an interface between experience and genome change.Radiation-induced bystander effects in cultured human stem cells.Role of oxidatively induced DNA lesions in human pathogenesis In situ biological dose mapping estimates the radiation burden delivered to 'spared' tissue between synchrotron X-ray microbeam radiotherapy tracks.Systemic DNA damage accumulation under in vivo tumor growth can be inhibited by the antioxidant Tempol Tumors induce complex DNA damage in distant proliferative tissues in vivo Long-distance inflammatory and genotoxic impact of cancer in vivo.Macrophages, nitric oxide and microRNAs are associated with DNA damage response pathway and senescence in inflammatory bowel disease Evaluation of the efficacy of radiation-modifying compounds using γH2AX as a molecular marker of DNA double-strand breaks Methyltransferases mediate cell memory of a genotoxic insult Para-inflammation mediates systemic DNA damage in response to tumor growth.Recent developments in the use of γ-H2AX as a quantitative DNA double-strand break biomarker.Systemic DNA damage related to cancer H2AX phosphorylation in response to DNA double-strand break formation during bystander signalling: effect of microRNA knockdown.Cytochrome-c mediated a bystander response dependent on inducible nitric oxide synthase in irradiated hepatoma cells.Modeling DNA double-strand break repair kinetics as an epiregulated cell-community-wide (epicellcom) response to radiation stress BRCA2 protein deficiency exaggerates doxorubicin-induced cardiomyocyte apoptosis and cardiac failure H2AX phosphorylation at the sites of DNA double-strand breaks in cultivated mammalian cells and tissues.Role of nitric oxide in the radiation-induced bystander effect.Human embryonic stem cell responses to ionizing radiation exposures: current state of knowledge and future challenges Susceptibility to bystander DNA damage is influenced by replication and transcriptional activity.Emerging roles of extracellular vesicles in the adaptive response of tumour cells to microenvironmental stress.DNA damage focus analysis in blood samples of minipigs reveals acute partial body irradiation.Silk-based materials for biomedical applications.Superoxide anion and hydrogen peroxide-induced signaling and damage in angiotensin II and aldosterone action.Cellular Senescence - its role in cancer and the response to ionizing radiation.The role of miRNA in the direct and indirect effects of ionizing radiation.Epigenetics in radiation-induced fibrosis.Human mesenchymal stem cells enhance the systemic effects of radiotherapy.DDR-mediated crosstalk between DNA-damaged cells and their microenvironment.Regulation of early signaling and gene expression in the alpha-particle and bystander response of IMR-90 human fibroblasts.A 4-Phenoxyphenol Derivative Exerts Inhibitory Effects on Human Hepatocellular Carcinoma Cells through Regulating Autophagy and Apoptosis Accompanied by Downregulating α-Tubulin Expression.Radioresistant human lung adenocarcinoma cells that survived multiple fractions of ionizing radiation are sensitive to HSP90 inhibition Radioprotection of targeted and bystander cells by methylproamine Bystander effect induced by UVC radiation in Chinese hamster V79 cells.

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Intercellular communication of cellular stress monitored by gamma-H2AX induction.

http://www.wikidata.org/entity/Q37376590

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 03 August 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@en

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@nl

type

label

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@en

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@nl

prefLabel

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@en

Intercellular communication of cellular stress monitored by gamma-H2AX induction.

@nl

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Intercellular communication of cellular stress monitored by gamma-H2AX induction.

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Brandon J Baird

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Christophe E Redon

http://www.w3.org/2001/XMLSchema#string

Jennifer S Dickey

http://www.w3.org/2001/XMLSchema#string

Mykyta V Sokolov

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Olga A Sedelnikova

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William M Bonner

http://www.w3.org/2001/XMLSchema#string

P2860

Both telomeric and non-telomeric DNA damage are determinants of mammalian cellular senescence

Germ-line mutations, DNA damage, and global hypermethylation in mice exposed to particulate air pollution in an urban/industrial location

Megabase chromatin domains involved in DNA double-strand breaks in vivo

Induction of sister chromatid exchanges by extremely low doses of alpha-particles

Oxidative damage is increased in human liver tissue adjacent to hepatocellular carcinoma

DNA double-strand breaks form in bystander cells after microbeam irradiation of three-dimensional human tissue models

PCNA, the maestro of the replication fork

GammaH2AX and cancer

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

Global gene expression analyses of bystander and alpha particle irradiated normal human lung fibroblasts: synchronous and differential responses.

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1686-1695

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scholarly article

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10.1093/CARCIN/BGP192

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10

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30

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2009-08-03T00:00:00Z

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26714112

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19651821

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2757548

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