Role of oxidatively induced DNA lesions in human pathogenesis - Wikidata - wikimedia - TriplyDB

Role of oxidatively induced DNA lesions in human pathogenesis

Role of oxidatively induced DNA lesions in human pathogenesis

http://www.wikidata.org/entity/Q34091785

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Variant base excision repair proteins: contributors to genomic instability Role of Redox Status in Development of Glioblastoma Association between OGG1 Ser326Cys polymorphism and risk of upper aero-digestive tract and gastrointestinal cancers: a meta-analysis Linking Pesticide Exposure with Pediatric Leukemia: Potential Underlying Mechanisms SOD2 deficiency promotes aging phenotypes in mouse skin Overview of base excision repair biochemistry DNA Damage Response and Autophagy: A Meaningful Partnership Discovery of direct inhibitors of Keap1-Nrf2 protein-protein interaction as potential therapeutic and preventive agents Positive Cofactor 4 (PC4) is critical for DNA repair pathway re-routing in DT40 cells Assessment of the mode of action underlying development of rodent small intestinal tumors following oral exposure to hexavalent chromium and relevance to humans Peto's Paradox: evolution's prescription for cancer prevention Homozygous mutation of MTPAP causes cellular radiosensitivity and persistent DNA double-strand breaks.Microbial pathogens trigger host DNA double-strand breaks whose abundance is reduced by plant defense responses.NADPH Oxidase Activation Contributes to Heavy Ion Irradiation-Induced Cell Death.The role of ROS and subsequent DNA-damage response in PUMA-induced apoptosis of ovarian cancer cells.In vivo and in vitro studies suggest a possible involvement of HPV infection in the early stage of breast carcinogenesis via APOBEC3B induction.DNA-PKcs deficiency leads to persistence of oxidatively induced clustered DNA lesions in human tumor cells.Telomere proteins POT1, TRF1 and TRF2 augment long-patch base excision repair in vitro.microRNAome changes in bystander three-dimensional human tissue models suggest priming of apoptotic pathways.Aging, cellular senescence, and cancer Systemic DNA damage accumulation under in vivo tumor growth can be inhibited by the antioxidant Tempol Structural/functional analysis of the human OXR1 protein: identification of exon 8 as the anti-oxidant encoding function.Small MAF genes variants and chronic myeloid leukemia.Oxidative DNA damage and nucleotide excision repair.The catalytic subunit of DNA-dependent protein kinase is required for cellular resistance to oxidative stress independent of DNA double-strand break repair.Viral carcinogenesis: factors inducing DNA damage and virus integration Defective repair of oxidative base lesions by the DNA glycosylase Nth1 associates with multiple telomere defects.Recent developments in the use of γ-H2AX as a quantitative DNA double-strand break biomarker.Oxidative stress-mediated regulation of proteasome complexes Unrepaired clustered DNA lesions induce chromosome breakage in human cells.Inhibition of oxidative metabolism leads to p53 genetic inactivation and transformation in neural stem cells.The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses.NADPH oxidase 4 is a critical mediator in Ataxia telangiectasia disease.Highly efficient conversion of superoxide to oxygen using hydrophilic carbon clusters.Oxidative stress in neurodegenerative diseases.Detection of clustered DNA lesions: Biological and clinical applications On the sequence-directed nature of human gene mutation: the role of genomic architecture and the local DNA sequence environment in mediating gene mutations underlying human inherited disease.Inactivation of NADPH oxidases NOX4 and NOX5 protects human primary fibroblasts from ionizing radiation-induced DNA damage.Induction of DNA damage in human urothelial cells by the brominated flame retardant 2,2-bis(bromomethyl)-1,3-propanediol: role of oxidative stress Dispersal evolution in neoplasms: the role of disregulated metabolism in the evolution of cell motility.

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Role of oxidatively induced DNA lesions in human pathogenesis

http://www.wikidata.org/entity/Q34091785

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年论文

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name

Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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Role of oxidatively induced DNA lesions in human pathogenesis

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J.MRREV.2009.12.005

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Mutation Research

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Role of oxidatively induced DNA lesions in human pathogenesis

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Alexandros G Georgakilas

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Asako J Nakamura

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Christophe E Redon

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Jennifer S Dickey

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Olga A Sedelnikova

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William M Bonner

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P2860

Both telomeric and non-telomeric DNA damage are determinants of mammalian cellular senescence

Free radicals and antioxidants in normal physiological functions and human disease

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

The phosphorylation status of PAS-B distinguishes HIF-1alpha from HIF-2alpha in NBS1 repression

Oncogenic bystander radiation effects in Patched heterozygous mouse cerebellum

Ataxia telangiectasia mutated activation by transcription- and topoisomerase I-induced DNA double-strand breaks

Mitochondrial DNA mutations in human disease

Processing of a complex multiply damaged DNA site by human cell extracts and purified repair proteins

Oxidative DNA damage: mechanisms, mutation, and disease

Exposing the MYtH about base excision repair and human inherited disease

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10.1016/J.MRREV.2009.12.005

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1-3

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20060490

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