Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats. - Wikidata - wikimedia - TriplyDB

Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats.

Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats.

http://www.wikidata.org/entity/Q37384183

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Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Prenatal Exposure to DEHP Affects Spermatogenesis and Sperm DNA Methylation in a Strain-Dependent Manner Endocrine-disrupting Chemicals: Review of Toxicological Mechanisms Using Molecular Pathway Analysis Medications as a potential source of exposure to phthalates among women of childbearing age Exposure to di(2-ethylhexyl) phthalate inhibits luteal function via dysregulation of CD31 and prostaglandin F2alpha in pregnant mice Toxicological mechanism of endocrine disrupting chemicals:is estrogen receptor involved?Sertoli-secreted FGF-2 induces PFKFB4 isozyme expression in mouse spermatogenic cells by activation of the MEK/ERK/CREB pathway.Altered expression of 3β-HSD, CYP17 and 17β-HSD in the foetal porcine gonads in response to anti-androgen flutamide exposure.Reproductive toxicity of di(2-ethylhexyl) phthalate in selenium-supplemented and selenium-deficient rats.Differential expression of connexin 43 in adult pig testes during normal spermatogenic cycle and after flutamide treatment.Flutamide alters the distribution of c-Src and affects the N-cadherin-β-catenin complex in the seminiferous epithelium of adult rat.

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Di-(2 ethylhexyl) phthalate and flutamide alter gene expression in the testis of immature male rats.

http://www.wikidata.org/entity/Q37384183

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 September 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Reproductive Biology and Endocrinology

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Di-(2 ethylhexyl) phthalate an ...... e testis of immature male rats

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Eui-Bae Jeung

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Eui-Man Jung

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Kyung-Chul Choi

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Thuy T B Vo

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Vu Hoang Dang

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Yeong-Min Yoo

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Differential modulation of Ca(v)2.1 channels by calmodulin and Ca2+-binding protein 1

Requirement of Lim1 for female reproductive tract development

Abnormalities of sexual development in male rats with in utero and lactational exposure to the antiandrogenic plasticizer Di(2-ethylhexyl) phthalate.

Normalization for cDNA microarray data: a robust composite method addressing single and multiple slide systematic variation.

The plasticizer diethylhexyl phthalate induces malformations by decreasing fetal testosterone synthesis during sexual differentiation in the male rat

Ca2+ binding protein-1 inhibits Ca2+ currents and exocytosis in bovine chromaffin cells

Novel role of phospholipase C-delta1: regulation of liver mitochondrial Ca2+ uptake

Correlation between protein accumulation profiles and conventional toxicological findings using a model antiandrogenic compound, flutamide

Testicular gene expression profiling following prepubertal rat mono-(2-ethylhexyl) phthalate exposure suggests a common initial genetic response at fetal and prepubertal ages

Identification of estrogen-regulated genes by microarray analysis of the uterus of immature rats exposed to endocrine disrupting chemicals

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