Role of chemokines in the enhancement of BBB permeability and inflammatory infiltration after rabies virus infection.
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IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous systemMonocytes and B cells support active replication of Chandipura virusNovel Approaches to the Prevention and Treatment of RabiesNoninvasive and targeted delivery of therapeutics to the brain using focused ultrasound.Intracerebral administration of recombinant rabies virus expressing GM-CSF prevents the development of rabies after infection with street virus.Expression of MIP-1alpha (CCL3) by a recombinant rabies virus enhances its immunogenicity by inducing innate immunity and recruiting dendritic cells and B cells.Enhancement of blood-brain barrier permeability is required for intravenously administered virus neutralizing antibodies to clear an established rabies virus infection from the brain and prevent the development of rabies in mice.Rabies virus expressing dendritic cell-activating molecules enhances the innate and adaptive immune response to vaccination.Met-CCL5 represents an immunotherapy strategy to ameliorate rabies virus infection.Expression of interferon gamma by a recombinant rabies virus strongly attenuates the pathogenicity of the virus via induction of type I interferon.Expression of neuronal CXCL10 induced by rabies virus infection initiates infiltration of inflammatory cells, production of chemokines and cytokines, and enhancement of blood-brain barrier permeability.Presence of virus neutralizing antibodies in cerebral spinal fluid correlates with non-lethal rabies in dogsComparison of the neuropathology induced by two West Nile virus strains.The inability of wild-type rabies virus to activate dendritic cells is dependent on the glycoprotein and correlates with its low level of the de novo-synthesized leader RNA.The relationship of CSF and plasma cytokine levels in HIV infected patients with neurocognitive impairment.Viral Infection of the Central Nervous System and Neuroinflammation Precede Blood-Brain Barrier Disruption during Japanese Encephalitis Virus Infection.Differential Host Immune Responses after Infection with Wild-Type or Lab-Attenuated Rabies Viruses in Dogs.Complete genome sequence of a street rabies virus from Mexico.Critical Role of K1685 and K1829 in the Large Protein of Rabies Virus in Viral Pathogenicity and Immune EvasionProfile of Cytokines and Chemokines Triggered by Wild-Type Strains of Rabies Virus in MiceRabies virus glycoprotein is an important determinant for the induction of innate immune responses and the pathogenic mechanisms.Role of the blood-brain barrier in rabies virus infection and protection.The roles of chemokines in rabies virus infection: overexpression may not always be beneficial.Phenotypic Consequences In vivo and In vitro of Rearranging the P Gene of RABV HEP-Flury.Enhancement of blood-brain barrier permeability and reduction of tight junction protein expression are modulated by chemokines/cytokines induced by rabies virus infection.Intrathecal humoral immunity to encephalitic RNA viruses.Immunological aspects of rabies: a literature review.Neuroinflammation and Infection: Molecular Mechanisms Associated with Dysfunction of Neurovascular Unit.Reverse genetics of rabies virus: new strategies to attenuate virus virulence for vaccine development.Characterization of street rabies virus variants with an additional N-glycan at position 247 in the glycoprotein.Wild-type rabies virus phosphoprotein is associated with viral sensitivity to type I interferon treatment.Rabies - epidemiology, pathogenesis, public health concerns and advances in diagnosis and control: a comprehensive review.Rabies vaccination at a virus-inoculated site as an alternative option to rabies immunoglobulin.Exogenous interferon prolongs survival of rabies infected mice.The CCL2-CCR2 system affects the progression and clearance of intracerebral hemorrhage.CXCL10 and blood-brain barrier modulation in rabies virus infection.Focused shockwave induced blood-brain barrier opening and transfection.Cerebrospinal fluid chemokine patterns in children with enterovirus 71-related encephalitis.Lab-Attenuated Rabies Virus Causes Abortive Infection and Induces Cytokine Expression in Astrocytes by Activating Mitochondrial Antiviral-Signaling Protein Signaling Pathway.
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Role of chemokines in the enhancement of BBB permeability and inflammatory infiltration after rabies virus infection.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 05 April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Role of chemokines in the enha ...... after rabies virus infection.
@en
Role of chemokines in the enha ...... after rabies virus infection.
@nl
type
label
Role of chemokines in the enha ...... after rabies virus infection.
@en
Role of chemokines in the enha ...... after rabies virus infection.
@nl
prefLabel
Role of chemokines in the enha ...... after rabies virus infection.
@en
Role of chemokines in the enha ...... after rabies virus infection.
@nl
P2093
P2860
P1433
P1476
Role of chemokines in the enha ...... after rabies virus infection.
@en
P2093
Sarah N Lackay
P2860
P356
10.1016/J.VIRUSRES.2009.03.014
P577
2009-04-05T00:00:00Z