Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
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Complement activation selectively potentiates the pathogenicity of the IgG2b and IgG3 isotypes of a high affinity anti-erythrocyte autoantibodyFc gamma R expression on macrophages is related to severity and chronicity of synovial inflammation and cartilage destruction during experimental immune-complex-mediated arthritis (ICA)Autoantibodies in systemic autoimmune diseases: specificity and pathogenicityDecreased NK Cell FcRgamma in HIV-1 infected individuals receiving combination antiretroviral therapy: a cross sectional studyMincle is an ITAM-coupled activating receptor that senses damaged cellsThe tyrosine phosphatase CD148 is an essential positive regulator of platelet activation and thrombosisCritical but overlapping role of FcgammaRIII and FcgammaRIV in activation of murine neutrophils by immobilized immune complexesLack of collagen XVIII/endostatin exacerbates immune-mediated glomerulonephritis.A role for adhesion and degranulation-promoting adapter protein in collagen-induced platelet activation mediated via integrin α(2) β(1)Ablation of a specific cell population by the replacement of a uniquely expressed gene with a toxin gene.Human neutrophil Fcgamma receptors initiate and play specialized nonredundant roles in antibody-mediated inflammatory diseasesGenetics of systemic autoimmunity in mouse models of lupus.Direct recognition of the mycobacterial glycolipid, trehalose dimycolate, by C-type lectin MincleApolipoprotein J/clusterin prevents a progressive glomerulopathy of agingNo evidence that soluble TACI induces signalling via membrane-expressed BAFF and APRIL in myeloid cells.Roles of Fc receptors in autoimmunity.IL-15, a survival factor for kidney epithelial cells, counteracts apoptosis and inflammation during nephritis.Generation of a unique small molecule peptidomimetic that neutralizes lupus autoantibody activity.Anti-T cell immunoglobulin and mucin domain-2 monoclonal antibody exacerbates collagen-induced arthritis by stimulating B cellsThe role of FcgammaRIIa as an inflammatory mediator in rheumatoid arthritis and systemic lupus erythematosus.Fcgamma receptors in the initiation and progression of systemic lupus erythematosus.Both Fcgamma receptor I and Fcgamma receptor III mediate disease in accelerated nephrotoxic nephritisRole of novel rat-specific Fc receptor in macrophage activation associated with crescentic glomerulonephritisAttenuation of Immune-Mediated Renal Injury by Telmisartan, an Angiotensin Receptor Blocker and a Selective PPAR-γ Activator.Targeting Syk as a treatment for allergic and autoimmune disorders.Murine membranous nephropathy: immunization with α3(IV) collagen fragment induces subepithelial immune complexes and FcγR-independent nephrotic syndrome.Pathogenic autoantibodies in lupus nephritis.Fc receptor targeting in the treatment of allergy, autoimmune diseases and cancer.Uptake of Leishmania major by dendritic cells is mediated by Fcgamma receptors and facilitates acquisition of protective immunityPathology and protection in nephrotoxic nephritis is determined by selective engagement of specific Fc receptors.Markedly different pathogenicity of four immunoglobulin G isotype-switch variants of an antierythrocyte autoantibody is based on their capacity to interact in vivo with the low-affinity Fcgamma receptor III.Poly(ADP-ribose) polymerase-1 regulates the progression of autoimmune nephritis in males by inducing necrotic cell death and modulating inflammation.FcgammaRIIB as a modulator of autoimmune disease susceptibility.Identification of a major GpVI-binding locus in human type III collagen.Fc gamma RIII and Fc gamma RIV are indispensable for acute glomerular inflammation induced by switch variant monoclonal antibodies.Noncanonical autophagy is required for type I interferon secretion in response to DNA-immune complexes.Increased expression of FcgammaRI/CD64 on circulating monocytes parallels ongoing inflammation and nephritis in lupus.Novel targets for immunotherapy in glomerulonephritis.Dectin-2 is a Syk-coupled pattern recognition receptor crucial for Th17 responses to fungal infection.Anti-C1q autoantibodies deposit in glomeruli but are only pathogenic in combination with glomerular C1q-containing immune complexes.
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Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1998
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@en
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@nl
type
label
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@en
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@nl
prefLabel
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@en
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis.
@nl
P2093
P2860
P356
P1476
Resistance of Fc receptor- deficient mice to fatal glomerulonephritis
@en
P2093
Karasawa A
Ledermann B
P2860
P304
P356
10.1172/JCI3256
P407
P50
P577
1998-09-01T00:00:00Z