Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
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Identification of brain nuclei implicated in cocaine-primed reinstatement of conditioned place preference: a behaviour dissociable from sensitizationAmphetamine and pseudoephedrine cross-tolerance measured by c-Fos protein expression in brains of chronically treated ratsMolecular neurobiology of addiction: what’s all the (Δ)FosB about?Addiction-related gene regulation: risks of exposure to cognitive enhancers vs. other psychostimulantsDeltaFosB: a sustained molecular switch for addictionReview. Transcriptional mechanisms of addiction: role of DeltaFosBDistinct patterns of DeltaFosB induction in brain by drugs of abusePsychomotor stimulant- and opiate-induced c-fos mRNA expression patterns in the rat forebrain: comparisons between acute drug treatment and a drug challenge in sensitized animalsD1 dopamine receptor activation of multiple transcription factor genes in rat striatumStriatal Fos expression is indicative of dopamine D1/D2 synergism and receptor supersensitivityNeuronal and behavioural abnormalities in striatal function in DARPP-32-mutant miceRepeated methamphetamine administration differentially alters fos expression in caudate-putamen patch and matrix compartments and nucleus accumbensCocaine activates Homer1 immediate early gene transcription in the mesocorticolimbic circuit: differential regulation by dopamine and glutamate signalingChronic methamphetamine exposure suppresses the striatal expression of members of multiple families of immediate early genes (IEGs) in the rat: normalization by an acute methamphetamine injectionAmphetamine alters Ras-guanine nucleotide-releasing factor expression in the rat striatum in vivoHistidine decarboxylase deficiency causes tourette syndrome: parallel findings in humans and miceAblation of rat TRPV1-expressing Adelta/C-fibers with resiniferatoxin: analysis of withdrawal behaviors, recovery of function and molecular correlates.The rate of intravenous cocaine administration alters c-fos mRNA expression and the temporal dynamics of dopamine, but not glutamate, overflow in the striatum.Regulation of immediate early gene expression and AP-1 binding in the rat nucleus accumbens by chronic cocaine.Relapse to cocaine seeking increases activity-regulated gene expression differentially in the prefrontal cortex of abstinent rats.Beyond Neuronal Activity Markers: Select Immediate Early Genes in Striatal Neuron Subtypes Functionally Mediate Psychostimulant Addiction.Increased activity of cyclin-dependent kinase 5 leads to attenuation of cocaine-mediated dopamine signalingCommon Hepatic Branch of Vagus Nerve-Dependent Expression of Immediate Early Genes in the Mouse Brain by Intraportal L-Arginine: Comparison with Cholecystokinin-8.Blockade of neurotensin receptors suppresses the dopamine D1/D2 synergism on immediate early gene expression in the rat brain.The dopaminergic hyper-responsiveness of the shell of the nucleus accumbens is hormone-dependent.Dopamine receptor genes: new tools for molecular psychiatry.Striatal regulation of ΔFosB, FosB, and cFos during cocaine self-administration and withdrawal.Interaction of the rostral basolateral amygdala and prelimbic prefrontal cortex in regulating reinstatement of cocaine-seeking behavior.Sphingosylphosphocholine, a signaling molecule which accumulates in Niemann-Pick disease type A, stimulates DNA-binding activity of the transcription activator protein AP-1.Region-specific induction of FosB/ΔFosB by voluntary alcohol intake: effects of naltrexone.Role of dorsal medial prefrontal cortex dopamine D1-family receptors in relapse to high-fat food seeking induced by the anxiogenic drug yohimbineGABA(B) receptor-positive modulation decreases selective molecular and behavioral effects of cocaineMotor-skill learning in a novel running-wheel task is dependent on D1 dopamine receptors in the striatum.Anatomical markers of activity in neuroendocrine systems: are we all 'fos-ed out'?Cocaine and the AP-1 transcription factor complex.Liquiritigenin decreases selective molecular and behavioral effects of cocaine in rodents.Leptin in the hindbrain facilitates phosphorylation of STAT3 in the hypothalamusRole of monoamine systems in activation of zif268 by cocaineA Genetic Mouse Model of Parkinson's Disease Shows Involuntary Movements and Increased Postsynaptic Sensitivity to Apomorphine.Preference for cocaine- versus pup-associated cues differentially activates neurons expressing either Fos or cocaine- and amphetamine-regulated transcript in lactating, maternal rodents
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Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@en
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@nl
type
label
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@en
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@nl
prefLabel
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@en
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@nl
P2093
P2860
P356
P1476
Cocaine induces striatal c-fos-immunoreactive proteins via dopaminergic D1 receptors.
@en
P2093
P2860
P304
P356
10.1073/PNAS.88.4.1291
P407
P577
1991-02-01T00:00:00Z