Long-lived Min mice develop advanced intestinal cancers through a genetically conservative pathway.
about
DNA-methylome analysis of mouse intestinal adenoma identifies a tumour-specific signature that is partly conserved in human colon cancerMouse models of colorectal cancer as preclinical modelsDynamic tumor growth patterns in a novel murine model of colorectal cancer.Discovery and validation of colonic tumor-associated proteins via metabolic labeling and stable isotopic dilution.Papilloma-pseudovirus eradicates intestinal tumours and triples the lifespan of ApcMin/+ mice.Gene expression profile and genomic alterations in colonic tumours induced by 1,2-dimethylhydrazine (DMH) in rats.Small animal micro-CT colonographyLongitudinal assessment of colonic tumor fate in mice by computed tomography and optical colonoscopyDistinguishing between cancer driver and passenger gene alteration candidates via cross-species comparison: a pilot study.Loss of Blm enhances basal cell carcinoma and rhabdomyosarcoma tumorigenesis in Ptch1+/- mice.Human cancer xenografts in outbred nude mice can be confounded by polymorphisms in a modifier of tumorigenesisDCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transitionAtlas of Wnt and R-spondin gene expression in the developing male mouse lower urogenital tract.Monoallelic silencing and haploinsufficiency in early murine intestinal neoplasms.Genetic Manipulation of Homologous Recombination In Vivo Attenuates Intestinal Tumorigenesis.Advanced Intestinal Cancers often Maintain a Multi-Ancestral Architecture.Colon Tumors with the Simultaneous Induction of Driver Mutations in APC, KRAS, and PIK3CA Still Progress through the Adenoma-to-carcinoma Sequence.More than two decades of Apc modeling in rodentsHeterochromatin Protein 1 Binding Protein 3 Expression as a Candidate Marker of Intrinsic 5-Fluorouracil Resistance.Ethanol-induced mast cell-mediated inflammation leads to increased susceptibility of intestinal tumorigenesis in the APC Δ468 min mouse model of colon cancerDclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells.Rodent intestinal epithelial carcinogenesis: pathology and preclinical models.Spontaneous initiation, promotion and progression of colorectal cancer in the novel A/J Min/+ mouse.Cecal microbiota association with tumor load in a colorectal cancer mouse model.Deficiency of caveolin-1 in Apc(min/+) mice promotes colorectal tumorigenesis.
P2860
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P2860
Long-lived Min mice develop advanced intestinal cancers through a genetically conservative pathway.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 07 July 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Long-lived Min mice develop ad ...... etically conservative pathway.
@en
Long-lived Min mice develop ad ...... etically conservative pathway.
@nl
type
label
Long-lived Min mice develop ad ...... etically conservative pathway.
@en
Long-lived Min mice develop ad ...... etically conservative pathway.
@nl
prefLabel
Long-lived Min mice develop ad ...... etically conservative pathway.
@en
Long-lived Min mice develop ad ...... etically conservative pathway.
@nl
P2093
P2860
P1433
P1476
Long-lived Min mice develop ad ...... etically conservative pathway.
@en
P2093
Alanna White
Amy J Prunuske
Donna G Albertson
Henry C Pitot
Jeffery W Bacher
Jesse Waggoner
Kristen Rasmussen
Linda Clipson
Mary Kay Washington
Richard B Halberg
P2860
P304
P356
10.1158/0008-5472.CAN-09-0446
P407
P50
P577
2009-07-07T00:00:00Z