Matrix metalloproteinase-7 regulates cleavage of pro-nerve growth factor and is neuroprotective following kainic acid-induced seizures.
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Matrix metalloproteinases in neural development: a phylogenetically diverse perspectiveMatrix metalloproteinases and minocycline: therapeutic avenues for fragile X syndrome.Multifaceted Roles of Metzincins in CNS Physiology and Pathology: From Synaptic Plasticity and Cognition to Neurodegenerative Disordersp75NTR, but not proNGF, is upregulated following status epilepticus in mice.Small-molecule modulation of neurotrophin receptors: a strategy for the treatment of neurological disease.LPS-induced proNGF synthesis and release in the N9 and BV2 microglial cells: a new pathway underling microglial toxicity in neuroinflammationDifferential regulation of macrophage phenotype by mature and pro-nerve growth factor.Pro-nerve growth factor in the ovary and human granulosa cells.Rapid Increases in proBDNF after Pilocarpine-Induced Status Epilepticus in Mice Are Associated with Reduced proBDNF Cleavage MachineryShaping neurons: Long and short range effects of mature and proBDNF signalling upon neuronal structure.Interleukin-1β enhances neuronal vulnerability to proNGF-mediated apoptosis by increasing surface expression of p75(NTR) and sortillin.Roles of matrix metalloproteinases and their targets in epileptogenesis and seizuresTime dependent integration of matrix metalloproteinases and their targeted substrates directs axonal sprouting and synaptogenesis following central nervous system injury.Dynamic nature of the p75 neurotrophin receptor in response to injury and diseaseFrom demyelination to remyelination: the road toward therapies for spinal cord injury.PROneurotrophins and CONSequences.Mir-592 regulates the induction and cell death-promoting activity of p75NTR in neuronal ischemic injury.Blockade of p75 Neurotrophin Receptor Reverses Irritability and Anxiety-Related Behaviors in a Rat Model of Status EpilepticusA Novel Neuroprotective Mechanism for Lithium That Prevents Association of the p75NTR-Sortilin Receptor Complex and Attenuates proNGF-Induced Neuronal Death In Vitro and In Vivo.Ghrelin ameliorates nerve growth factor Dysmetabolism and inflammation in STZ-induced diabetic rats.ProBDNF/p75NTR/sortilin pathway is activated in peripheral blood of patients with alcohol dependence.Matrix Metalloproteinase-Mediated Blood-Brain Barrier Dysfunction in Epilepsy.The pharmacological perturbation of brain zinc impairs BDNF-related signaling and the cognitive performances of young mice.
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P2860
Matrix metalloproteinase-7 regulates cleavage of pro-nerve growth factor and is neuroprotective following kainic acid-induced seizures.
description
article científic
@ca
article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on January 2012
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@en
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@nl
type
label
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@en
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@nl
prefLabel
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@en
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@nl
P2860
P1476
Matrix metalloproteinase-7 reg ...... kainic acid-induced seizures.
@en
P2093
Audrey P Le
Wilma J Friedman
P2860
P304
P356
10.1523/JNEUROSCI.4128-11.2012
P407
P577
2012-01-01T00:00:00Z