Skeletal muscle AMP-activated protein kinase is essential for the metabolic response to exercise in vivo.
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Regulation of glucose and glycogen metabolism during and after exerciseRegulation of exercise-stimulated glucose uptake in skeletal muscleExercise Modulates Oxidative Stress and Inflammation in Aging and Cardiovascular DiseasesSkeletal muscle nitric oxide signaling and exercise: a focus on glucose metabolismExperimental studies of the molecular pathways regulated by exercise and resveratrol in heart, skeletal muscle and the vasculatureStatin-Induced Increases in Atrophy Gene Expression Occur Independently of Changes in PGC1α Protein and Mitochondrial ContentCarbon monoxide improves cardiac function and mitochondrial population quality in a mouse model of metabolic syndromeContraction-stimulated glucose transport in muscle is controlled by AMPK and mechanical stress but not sarcoplasmatic reticulum Ca(2+) releaseTwo weeks of metformin treatment enhances mitochondrial respiration in skeletal muscle of AMPK kinase dead but not wild type miceKinetics of contraction-induced GLUT4 translocation in skeletal muscle fibers from living mice.How to explain exercise-induced phenotype from molecular data: rethink and reconstruction based on AMPK and mTOR signaling.Activating HSP72 in rodent skeletal muscle increases mitochondrial number and oxidative capacity and decreases insulin resistance.Disruption of BCAA metabolism in mice impairs exercise metabolism and endurance.AMP-activated protein kinase (AMPK)α2 plays a role in determining the cellular fate of glucose in insulin-resistant mouse skeletal muscle.Endothelial nitric oxide synthase is central to skeletal muscle metabolic regulation and enzymatic signaling during exercise in vivo.Activation of AMP-activated protein kinase is required for berberine-induced reduction of atherosclerosis in mice: the role of uncoupling protein 2.Tissue depletion of taurine accelerates skeletal muscle senescence and leads to early death in mice.Whole body deletion of AMP-activated protein kinase {beta}2 reduces muscle AMPK activity and exercise capacity.Tissue taurine depletion alters metabolic response to exercise and reduces running capacity in mice.Obesity impairs skeletal muscle AMPK signaling during exercise: role of AMPKα2 in the regulation of exercise capacity in vivo.Inhibition of the AMP-activated protein kinase-α2 accentuates agonist-induced vascular smooth muscle contraction and high blood pressure in mice.AMP-activated protein kinase (AMPK) beta1beta2 muscle null mice reveal an essential role for AMPK in maintaining mitochondrial content and glucose uptake during exercise.RETRACTED: Hepatic glucagon action is essential for exercise-induced reversal of mouse fatty liver.Improved Muscle Function in Duchenne Muscular Dystrophy through L-Arginine and Metformin: An Investigator-Initiated, Open-Label, Single-Center, Proof-Of-Concept-Study.Mesenchymal stem cell transplantation for the infarcted heart: a role in minimizing abnormalities in cardiac-specific energy metabolism.Tbx15 controls skeletal muscle fibre-type determination and muscle metabolism.Mitochondrial antioxidative capacity regulates muscle glucose uptake in the conscious mouse: effect of exercise and diet.Overexpression of sphingosine kinase 1 prevents ceramide accumulation and ameliorates muscle insulin resistance in high-fat diet-fed mice.AMPK and Exercise: Glucose Uptake and Insulin Sensitivity.LKB1 regulates lipid oxidation during exercise independently of AMPKHyaluronan accumulates with high-fat feeding and contributes to insulin resistance.Relaxin treatment reverses insulin resistance in mice fed a high-fat dietThe IL-6 Paradox: Context Dependent Interplay of SOCS3 and AMPK.α1AMP-activated protein kinase mediates vascular protective effects of exercise.Muscle-specific AMPK β1β2-null mice display a myopathy due to loss of capillary density in nonpostural muscles.AMP-activated protein kinase and metabolic control.Contraction-induced signaling: evidence of convergent cascades in the regulation of muscle fatty acid metabolism.Role of nitric oxide in skeletal muscle glucose uptake during exercise.Activation of AMPK and its Impact on Exercise Capacity.Skeletal myofiber vascular endothelial growth factor is required for the exercise training-induced increase in dentate gyrus neuronal precursor cells.
P2860
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P2860
Skeletal muscle AMP-activated protein kinase is essential for the metabolic response to exercise in vivo.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 12 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@en
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@nl
type
label
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@en
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@nl
prefLabel
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@en
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@nl
P2093
P2860
P356
P1476
Skeletal muscle AMP-activated ...... response to exercise in vivo.
@en
P2093
David H Wasserman
Deanna P Bracy
Julio E Ayala
Owen P McGuinness
Robert S Lee-Young
Sara E Lynes
Susan R Griffee
P2860
P304
23925-23934
P356
10.1074/JBC.M109.021048
P407
P577
2009-06-12T00:00:00Z