The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
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MicroRNA-200 family members differentially regulate morphological plasticity and mode of melanoma cell invasionThe ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?MicroRNAs and metastasis: little RNAs go a long wayThe miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identityThe microRNA-200 family: small molecules with novel roles in cancer development, progression and therapymiR-8 modulates cytoskeletal regulators to influence cell survival and epithelial organization in Drosophila wings.Repression of choroidal neovascularization through actin cytoskeleton pathways by microRNA-24.The real-time dynamic monitoring of microRNA function in cholangiocarcinoma.A novel pathway of TEF regulation mediated by microRNA-125b contributes to the control of actin distribution and cell shape in fibroblasts.MicroRNA-200b suppresses arsenic-transformed cell migration by targeting protein kinase Cα and Wnt5b-protein kinase Cα positive feedback loop and inhibiting Rac1 activationThe role of the miR-200 family in epithelial-mesenchymal transition.MicroRNA-200b targets protein kinase Cα and suppresses triple-negative breast cancer metastasisOverexpression of microRNA-200c predicts poor outcome in patients with PR-negative breast cancer.Surfing the big WAVE: Insights into the role of WAVE3 as a driving force in cancer progression and metastasisIncreased expression levels of WAVE3 are associated with the progression and metastasis of triple negative breast cancer.Global protein conjugation by ubiquitin-like-modifiers during ischemic stress is regulated by microRNAs and confers robust tolerance to ischemia.MicroRNA-200 family modulation in distinct breast cancer phenotypes.miR-200c targets a NF-κB up-regulated TrkB/NTF3 autocrine signaling loop to enhance anoikis sensitivity in triple negative breast cancerWAVE3, an actin remodeling protein, is regulated by the metastasis suppressor microRNA, miR-31, during the invasion-metastasis cascade.WAVE3-NFκB interplay is essential for the survival and invasion of cancer cells.Targets of miR-200c mediate suppression of cell motility and anoikis resistancemiR-31 is a broad regulator of β1-integrin expression and function in cancer cells.MicroRNA-200c represses migration and invasion of breast cancer cells by targeting actin-regulatory proteins FHOD1 and PPM1F.miR-31 and its host gene lncRNA LOC554202 are regulated by promoter hypermethylation in triple-negative breast cancer.MicroRNA-34a modulates cytoskeletal dynamics through regulating RhoA/Rac1 cross-talk in chondroblasts.Mechanistic insights into the role of microRNAs in cancer: influence of nutrient crosstalk.MiR-200b regulates autophagy associated with chemoresistance in human lung adenocarcinomaLoss of miR-200b promotes invasion via activating the Kindlin-2/integrin β1/AKT pathway in esophageal squamous cell carcinoma: An E-cadherin-independent mechanismMicroRNA-200b is overexpressed in endometrial adenocarcinomas and enhances MMP2 activity by downregulating TIMP2 in human endometrial cancer cell line HEC-1A cells.miR141 expression is downregulated and negatively correlated with STAT5 expression in esophageal squamous cell carcinoma.Actin-binding protein regulation by microRNAs as a novel microbial strategy to modulate phagocytosis by host cells: the case of N-Wasp and miR-142-3pUpregulated WAVE3 expression is essential for TGF-β-mediated EMT and metastasis of triple-negative breast cancer cells.Kindlin-3 enhances breast cancer progression and metastasis by activating Twist-mediated angiogenesis.MicroRNAs (miRNAs) in cancer invasion and metastasis: therapeutic approaches based on metastasis-related miRNAs.Actin binding proteins: their ups and downs in metastatic life.SUMO and ischemic tolerance.The involvement of JAK-STAT3 in cell motility, invasion, and metastasis.miR-138-Mediated Regulation of KINDLIN-2 Expression Modulates Sensitivity to Chemotherapeutics.A core microRNA signature associated with inducers of the epithelial-to-mesenchymal transition.MiR-200 can repress breast cancer metastasis through ZEB1-independent but moesin-dependent pathways.
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P2860
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on October 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
@en
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion.
@nl
type
label
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
@en
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion.
@nl
prefLabel
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
@en
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion.
@nl
P2093
P2860
P356
P1476
The miR200 family of microRNAs regulates WAVE3-dependent cancer cell invasion
@en
P2093
Edward F Plow
Katarzyna Bialkowska
Khalid Sossey-Alaoui
P2860
P304
33019-33029
P356
10.1074/JBC.M109.034553
P407
P577
2009-10-01T00:00:00Z