Helminth infection can reduce insulitis and type 1 diabetes through CD25- and IL-10-independent mechanisms. - Wikidata - wikimedia - TriplyDB

Helminth infection can reduce insulitis and type 1 diabetes through CD25- and IL-10-independent mechanisms.

Helminth infection can reduce insulitis and type 1 diabetes through CD25- and IL-10-independent mechanisms.

http://www.wikidata.org/entity/Q37451115

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Diplomatic Assistance: Can Helminth-Modulated Macrophages Act as Treatment for Inflammatory Disease?Helminth infections and host immune regulation Alternatively activated macrophages in types 1 and 2 diabetes Understanding the role of antibodies in murine infections with Heligmosomoides (polygyrus) bakeri: 35 years ago, now and 35 years ahead Unraveling the Hygiene Hypothesis of helminthes and autoimmunity: origins, pathophysiology, and clinical applications Helminthic therapy: improving mucosal barrier function Immunity to the model intestinal helminth parasite Heligmosomoides polygyrus Proteomic analysis of excretory-secretory products of Heligmosomoides polygyrus assessed with next-generation sequencing transcriptomic information Concise Review: Macrophages: Versatile Gatekeepers During Pancreatic β-Cell Development, Injury, and Regeneration.Helminth Immunomodulation in Autoimmune Disease.Helminth-host immunological interactions: prevention and control of immune-mediated diseases Improving power of genome-wide association studies with weighted false discovery rate control and prioritized subset analysis.Helminth secretome database (HSD): a collection of helminth excretory/secretory proteins predicted from expressed sequence tags (ESTs).Decreased prevalence of lymphatic filariasis among subjects with type-1 diabetes.Secreted proteins from the helminth Fasciola hepatica inhibit the initiation of autoreactive T cell responses and prevent diabetes in the NOD mouse Helminth infections decrease host susceptibility to immune-mediated diseases.Immune regulators of inflammation in obesity-associated type 2 diabetes and coronary artery disease.Differential changes in expression of intestinal antimicrobial peptide genes during Ascaris lumbricoides infection in Zambian adults do not respond to helminth eradication.Systemic impact of intestinal helminth infections.Beyond genetics. Influence of dietary factors and gut microbiota on type 1 diabetes.Immunological Basis for Rapid Progression of Diabetes in Older NOD Mouse Recipients Post BM-HSC Transplantation Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.Parasitic helminths and their beneficial impact on type 1 and type 2 diabetes.Nematode-bacterium symbioses--cooperation and conflict revealed in the "omics" age.Chronic Gastrointestinal Nematode Infection Mutes Immune Responses to Mycobacterial Infection Distal to the Gut.Proteomic analysis of secretory products from the model gastrointestinal nematode Heligmosomoides polygyrus reveals dominance of venom allergen-like (VAL) proteins Parasitic nematode-induced modulation of body weight and associated metabolic dysfunction in mouse models of obesity.The hygiene hypothesis: current perspectives and future therapies Helminth infection and type 1 diabetes.Role of macrophages in the altered epithelial function during a type 2 immune response induced by enteric nematode infection.Parasitic helminths: new weapons against immunological disorders.Helminth infections: therapeutic potential in autoimmune disorders.Prevention or acceleration of type 1 diabetes by viruses.Trichinella spiralis immunomodulation: an interactive multifactorial process.Induction of regulatory cells by helminth parasites: exploitation for the treatment of inflammatory diseases.Helminths in the hygiene hypothesis: sooner or later?Mast cells: new therapeutic target in helminth immune modulation.Therapeutic potential of the immunomodulatory proteins Wuchereria bancrofti L2 and Brugia malayi abundant larval transcript 2 against streptozotocin-induced type 1 diabetes in mice.Immunomodulation by helminths: Similar impact on type 1 and type 2 diabetes?Therapeutic potential of helminths in autoimmune diseases: helminth-derived immune-regulators and immune balance.

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Helminth infection can reduce insulitis and type 1 diabetes through CD25- and IL-10-independent mechanisms.

http://www.wikidata.org/entity/Q37451115

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scientific article published on 14 September 2009

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Helminth infection can reduce ...... IL-10-independent mechanisms.

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Helminth infection can reduce ...... IL-10-independent mechanisms.

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Helminth infection can reduce ...... IL-10-independent mechanisms.

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Infection and Immunity

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Helminth infection can reduce ...... IL-10-independent mechanisms.

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Andrew Gaydo

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David Bleich

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David Lagunoff

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Farhang Alem

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Gity Mousavi

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Pankaj K Mishra

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Qian Liu

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William C Gause

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Zhugong Liu

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P2860

Inhibition of autoimmune type 1 diabetes by gastrointestinal helminth infection

IL-10-dependent infectious tolerance after the treatment of experimental allergic encephalomyelitis with redirected CD4+CD25+ T lymphocytes

Adeno-associated virus vector-mediated IL-10 gene delivery prevents type 1 diabetes in NOD mice.

Chronic helminth infection induces alternatively activated macrophages expressing high levels of CCR5 with low interleukin-12 production and Th2-biasing ability.

Helminth parasites--masters of regulation.

IL-2 and autocrine IL-4 drive the in vivo development of antigen-specific Th2 T cells elicited by nematode parasites.

Infections and autoimmunity--good or bad?

Mucosal immunity and allergic responses: lack of regulation and/or lack of microbial stimulation?

Rising incidence of insulin dependent diabetes in children aged under 5 years in the Oxford region: time trend analysis. The Bart's-Oxford Study Group.

Regulatory T cells induced by parasites and the modulation of allergic responses.

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5347-5358

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scholarly article

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10.1128/IAI.01170-08

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12

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77

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Krishnan Sundar

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2009-09-14T00:00:00Z

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26810052

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19752032

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type-1 diabetes

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2786463

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