Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. - Wikidata - wikimedia - TriplyDB

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.

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Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis Epstein-Barr virus latent genes The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1 Epstein-Barr virus nuclear antigen 3A protein regulates CDKN2B transcription via interaction with MIZ-1 Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.Epstein-Barr virus transcription factor Zta acts through distal regulatory elements to directly control cellular gene expression.Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells Epstein-Barr virus oncoprotein super-enhancers control B cell growth Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs.Epstein-Barr virus super-enhancer eRNAs are essential for MYC oncogene expression and lymphoblast proliferation IRF4 and IRF8: Governing the virtues of B Lymphocytes.The RUNX complex: reaching beyond haematopoiesis into immunity.Epstein-Barr virus latency: current and future perspectives.EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.Nonstructural Protein 4 of Porcine Reproductive and Respiratory Syndrome Virus Modulates Cell Surface Swine Leukocyte Antigen Class I Expression by Downregulating β2-Microglobulin Transcription.Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression RUNX super-enhancer control through the Notch pathway by Epstein-Barr virus transcription factors regulates B cell growth.SPIB and BATF provide alternate determinants of IRF4 occupancy in diffuse large B-cell lymphoma linked to disease heterogeneity.Epstein-Barr Virus LMP1 Mediated Oncogenicity.Regulation of tumour related genes by dynamic epigenetic alteration at enhancer regions in gastric epithelial cells infected by Epstein-Barr virus.Roles of RUNX in B Cell Immortalisation.The Use of Chromatin Precipitation Coupled to DNA Sequencing (ChIP-Seq) for the Analysis of Zta Binding to the Human and EBV Genome.The Epstein-Barr Virus Regulome in Lymphoblastoid Cells.Prevalence of targetable oncogenic mutations and genomic alterations in Epstein-Barr virus-associated diffuse large B-cell lymphoma of the elderly.Integrative Analysis of Transcription Factor Combinatorial Interactions Using a Bayesian Tensor Factorization Approach.Epstein-Barr Virus Induces Adhesion Receptor CD226 (DNAM-1) Expression during Primary B-Cell Transformation into Lymphoblastoid Cell Lines.EBV and Apoptosis: The Viral Master Regulator of Cell Fate?CRISPR-Cas9 Genetic Analysis of Virus-Host Interactions.Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.Current Progress in EBV-Associated B-Cell Lymphomas.Epstein-Barr virus nuclear antigen EBNA-LP is essential for transforming naïve B cells, and facilitates recruitment of transcription factors to the viral genome.The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.Transcriptional and epigenetic modulation of autophagy promotes EBV oncoprotein EBNA3C induced B-cell survival.Identification of Novel Functional Variants of SIN3A and SRSF1 among Somatic Variants in Acute Myeloid Leukemia Patients.Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of and the Locus via Interaction with the Histone Lysine Demethylase KDM2B Epstein-Barr Virus-Associated Malignancies: Roles of Viral Oncoproteins in Carcinogenesis

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Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.

http://www.wikidata.org/entity/Q37474974

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 16 December 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

@en

Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

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type

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Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

@en

Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

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prefLabel

Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

@en

Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

@nl

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PNAS.1321704111

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Proceedings of the National Academy of Sciences of the United States of America

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Epstein-Barr virus nuclear ant ...... ruits Sin3A to repress CDKN2A.

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Amy M Holthaus

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Anqi Wang

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Bo Zhao

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Bradford Willox

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Eric C Johannsen

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Hufeng Zhou

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Peter V Kharchenko

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Seiji Maruo

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Sizun Jiang

http://www.w3.org/2001/XMLSchema#string

Tommy T Shi

http://www.w3.org/2001/XMLSchema#string

P2860

Ultrafast and memory-efficient alignment of short DNA sequences to the human genome

A role for SKIP in EBNA2 activation of CBF1-repressed promoters

Epstein-Barr virus latent antigen 3C can mediate the degradation of the retinoblastoma protein through an SCF cellular ubiquitin ligase

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2.

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

A conserved domain of the Epstein-Barr virus nuclear antigens 3A and 3C binds to a discrete domain of Jkappa

Genome-wide mapping of HATs and HDACs reveals distinct functions in active and inactive genes

ChIP-seq guidelines and practices of the ENCODE and modENCODE consortia

A genomic regulatory element that directs assembly and function of immune-specific AP-1-IRF complexes

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421-426

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scholarly article

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10.1073/PNAS.1321704111

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1

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111

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Elliott D. Kieff

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2013-12-16T00:00:00Z

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259350639

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24344258

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Epstein–Barr virus

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3890834

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